Touzet Pascal, Meyer Etienne H
Laboratoire Génétique et Evolution des Populations Végétales, CNRS UMR 8198, Université Lille 1, F-59655 Villeneuve d'Ascq cedex, France.
Max Planck Institute of Molecular Plant Physiology, Am Mühlenberg 1, D-14476 Potsdam-Golm, Germany.
Mitochondrion. 2014 Nov;19 Pt B:166-71. doi: 10.1016/j.mito.2014.04.009. Epub 2014 Apr 24.
Cytoplasmic male sterility (CMS) is a common feature encountered in plant species. It is the result of a genomic conflict between the mitochondrial and the nuclear genomes. CMS is caused by mitochondrial encoded factors which can be counteracted by nuclear encoded factors restoring male fertility. Despite extensive work, the molecular mechanism of male sterility still remains unknown. Several studies have suggested the involvement of respiration on the disruption of pollen production through an energy deficiency. By comparing recent works on CMS and respiratory mutants, we suggest that the "ATP hypothesis" might not be as obvious as previously suggested.
细胞质雄性不育（CMS）是植物物种中常见的一种特征。它是线粒体基因组与核基因组之间基因组冲突的结果。CMS由线粒体编码的因子引起，而核编码的因子可以抵消这些因子，从而恢复雄性育性。尽管进行了大量研究，但雄性不育的分子机制仍然未知。一些研究表明，呼吸作用通过能量缺乏参与了花粉生产的破坏过程。通过比较最近关于CMS和呼吸突变体的研究，我们认为“ATP假说”可能并不像之前所认为的那么明显。
