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对与年龄相关的死亡率上升速率中黑/白差异的一项研究。

An examination of black/white differences in the rate of age-related mortality increase.

作者信息

Fenelon Andrew

机构信息

Brown University.

出版信息

Demogr Res. 2013 Jul;29:441-472. doi: 10.4054/DemRes.2013.29.17.

Abstract

BACKGROUND

The rate of mortality increase with age among adults is typically used as a measure of the rate of functional decline associated with aging or senescence. While black and white populations differ in the level of mortality, mortality also rises less rapidly with age for blacks than for whites, leading to the well-known black/white mortality "crossover".

OBJECTIVE

This paper investigates black/white differences in the rate of mortality increase with age for major causes of death in order to examine the factors responsible for the black/white crossover.

METHODS

The analysis considers two explanations for the crossover: and . Mortality is modeled using a Gompertz model for 11 causes of death from ages 50-84 among blacks and whites by sex.

RESULTS

Mortality increases more rapidly with age for whites than for blacks for nearly all causes of death considered. The all-cause mortality rate of mortality increase is nearly two percentage points higher for whites. The analysis finds evidence for both selective survival and age misreporting, although age misreporting is a more prominent explanation among women.

CONCLUSIONS

The black/white mortality crossover reflects large differences in the rate of age-related mortality increase. Instead of reflecting the impact of specific causes of death, this pattern exists across many disparate disease conditions, indicating the need for a broad explanation.

摘要

背景

成年人中死亡率随年龄增长的速率通常被用作衡量与衰老或老年化相关的功能衰退速率的指标。虽然黑人和白人在死亡率水平上存在差异,但黑人死亡率随年龄增长的速度也比白人慢,从而导致了著名的黑/白死亡率“交叉”现象。

目的

本文研究主要死因的死亡率随年龄增长速率的黑/白差异,以探讨导致黑/白交叉现象的因素。

方法

该分析考虑了交叉现象的两种解释: 和 。使用Gompertz模型对50至84岁黑人和白人按性别划分的11种死因的死亡率进行建模。

结果

在所考虑的几乎所有死因中,白人死亡率随年龄增长的速度都比黑人快。白人死亡率增长的全因死亡率几乎高出两个百分点。分析发现了选择性生存和年龄误报的证据,尽管年龄误报在女性中是一个更突出的解释。

结论

黑/白死亡率交叉反映了与年龄相关的死亡率增长速率的巨大差异。这种模式并非反映特定死因的影响,而是存在于许多不同的疾病状况中,这表明需要一个广泛的解释。

https://cdn.ncbi.nlm.nih.gov/pmc/blobs/b3aa/3998199/d5ff4a21ce7e/nihms571405f1.jpg

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