白细胞介素-35采用“B”途径。 - Suppr

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超能文献

Interleukin-35 takes the 'B' line.

作者信息

Mauri Claudia, Nistala Kiran

机构信息

Centre for Rheumatology, Division of Medicine, University College London, London, UK.

出版信息

Nat Med. 2014 Jun;20(6):580-1. doi: 10.1038/nm.3594.

DOI:10.1038/nm.3594

PMID:24901562

Abstract

摘要

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本文引用的文献

Interleukin-35 induces regulatory B cells that suppress autoimmune disease.白细胞介素-35 诱导调节性 B 细胞抑制自身免疫性疾病。

Nat Med. 2014 Jun;20(6):633-41. doi: 10.1038/nm.3554. Epub 2014 Apr 17.

IL-35-producing B cells are critical regulators of immunity during autoimmune and infectious diseases.产生白细胞介素-35的B细胞是自身免疫性疾病和感染性疾病期间免疫的关键调节因子。

Nature. 2014 Mar 20;507(7492):366-370. doi: 10.1038/nature12979. Epub 2014 Feb 23.

Trypanosoma cruzi trans-sialidase initiates a program independent of the transcription factors RORγt and Ahr that leads to IL-17 production by activated B cells.克氏锥虫 trans-sialidase 启动了一个独立于转录因子 RORγt 和 Ahr 的程序，导致激活的 B 细胞产生 IL-17。

Nat Immunol. 2013 May;14(5):514-22. doi: 10.1038/ni.2569. Epub 2013 Apr 7.

CD19+CD24hiCD38hi B cells maintain regulatory T cells while limiting TH1 and TH17 differentiation.CD19+CD24hiCD38hi B 细胞在维持调节性 T 细胞的同时限制 TH1 和 TH17 细胞的分化。

Sci Transl Med. 2013 Feb 20;5(173):173ra23. doi: 10.1126/scitranslmed.3005407.

Regulatory B cells control T-cell autoimmunity through IL-21-dependent cognate interactions.调节性 B 细胞通过 IL-21 依赖的同源相互作用来控制 T 细胞自身免疫。

Nature. 2012 Nov 8;491(7423):264-8. doi: 10.1038/nature11501. Epub 2012 Oct 14.

The composition and signaling of the IL-35 receptor are unconventional.IL-35 受体的组成和信号传导是非传统的。

Nat Immunol. 2012 Feb 5;13(3):290-9. doi: 10.1038/ni.2227.

Immune regulatory function of B cells.B 细胞的免疫调节功能。

Annu Rev Immunol. 2012;30:221-41. doi: 10.1146/annurev-immunol-020711-074934. Epub 2012 Jan 3.

IL-12p35 subunit contributes to autoimmunity by limiting IL-27-driven regulatory responses.IL-12p35 亚基通过限制 IL-27 驱动的调节反应而有助于自身免疫。

J Immunol. 2011 Sep 15;187(6):3402-12. doi: 10.4049/jimmunol.1100224. Epub 2011 Aug 15.

Regulatory B cells are identified by expression of TIM-1 and can be induced through TIM-1 ligation to promote tolerance in mice.调节性 B 细胞通过 TIM-1 的表达来鉴定，并且可以通过 TIM-1 的连接来诱导，以促进小鼠的耐受。

J Clin Invest. 2011 Sep;121(9):3645-56. doi: 10.1172/JCI46274. Epub 2011 Aug 8.

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