关于“旁路途径功能不需要MASP - 1/-3的前因子D裂解活性”的评论 - Suppr

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超能文献

Comment on "the pro-factor D cleaving activity of MASP-1/-3 is not required for alternative pathway function".

作者信息

Takahashi Minoru, Sekine Hideharu, Fujita Teizo

机构信息

Department of Immunology, Fukushima Medical University School of Medicine, Fukushima 960-1295, Japan

Department of Immunology, Fukushima Medical University School of Medicine, Fukushima 960-1295, Japan.

出版信息

J Immunol. 2014 Jun 15;192(12):5448-9. doi: 10.4049/jimmunol.1400766.

DOI:10.4049/jimmunol.1400766

PMID:24907377

Abstract

摘要

相似文献

Comment on "the pro-factor D cleaving activity of MASP-1/-3 is not required for alternative pathway function".关于“旁路途径功能不需要MASP - 1/-3的前因子D裂解活性”的评论

J Immunol. 2014 Jun 15;192(12):5448-9. doi: 10.4049/jimmunol.1400766.

The pro-factor D cleaving activity of MASP-1/-3 is not required for alternative pathway function.旁路途径功能并不需要MASP-1/-3的前因子D裂解活性。

J Immunol. 2014 Jun 15;192(12):5447-8. doi: 10.4049/jimmunol.1400777.

Comment on "Mannan-binding lectin-associated serine protease (MASP)-1 is crucial for lectin pathway activation in human serum, whereas neither MASP-1 nor MASP-3 is required for alternative pathway function".关于《甘露聚糖结合凝集素相关丝氨酸蛋白酶（MASP）-1对人血清中凝集素途径激活至关重要，而替代途径功能既不需要MASP-1也不需要MASP-3》的评论

J Immunol. 2013 Mar 15;190(6):2477. doi: 10.4049/jimmunol.1390002.

Response to comment on "Mannan-binding lectin-associated serine protease (MASP)-1 is crucial for lectin pathway activation in human serum, whereas neither MASP-1 nor MASP-3 is required for alternative pathway function".对“甘露聚糖结合凝集素相关丝氨酸蛋白酶（MASP）-1对人血清中凝集素途径的激活至关重要，而替代途径功能既不需要MASP-1也不需要MASP-3”一文评论的回应

J Immunol. 2013 Mar 15;190(6):2477-8. doi: 10.4049/jimmunol.1390003.

Mannan-binding lectin-associated serine protease (MASP)-1 is crucial for lectin pathway activation in human serum, whereas neither MASP-1 nor MASP-3 is required for alternative pathway function.甘露聚糖结合凝集素相关丝氨酸蛋白酶（MASP）-1 对人血清中凝集素途径的激活至关重要，而 MASP-1 和 MASP-3 均不参与替代途径的功能。

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Extensive Basal Level Activation of Complement Mannose-Binding Lectin-Associated Serine Protease-3: Kinetic Modeling of Lectin Pathway Activation Provides Possible Mechanism.补体甘露糖结合凝集素相关丝氨酸蛋白酶-3的广泛基础水平激活：凝集素途径激活的动力学模型提供了可能的机制。

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Mannan-Binding Lectin-Associated Serine Protease 1/3 Cleavage of Pro-Factor D into Factor D In Vivo and Attenuation of Collagen Antibody-Induced Arthritis through Their Targeted Inhibition by RNA Interference-Mediated Gene Silencing.甘露糖结合凝集素相关丝氨酸蛋白酶1/3在体内将前因子D裂解为因子D，并通过RNA干扰介导的基因沉默对其进行靶向抑制，从而减轻胶原抗体诱导的关节炎。

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MASP-3 is the exclusive pro-factor D activator in resting blood: the lectin and the alternative complement pathways are fundamentally linked.MASP-3 是静息血液中唯一的因子 D 前体激活物：凝集素和替代补体途径从根本上是相关联的。

Sci Rep. 2016 Aug 18;6:31877. doi: 10.1038/srep31877.

Novel MASP1 mutations are associated with an expanded phenotype in 3MC1 syndrome.新型MASP1突变与3MC1综合征的扩展表型相关。

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Essential role for the lectin pathway in collagen antibody-induced arthritis revealed through use of adenovirus programming complement inhibitor MAp44 expression.通过使用腺病毒编程补体抑制剂MAp44表达揭示凝集素途径在胶原抗体诱导的关节炎中的重要作用。

J Immunol. 2014 Sep 1;193(5):2455-68. doi: 10.4049/jimmunol.1400752. Epub 2014 Jul 28.

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