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Comment on "Mannan-binding lectin-associated serine protease (MASP)-1 is crucial for lectin pathway activation in human serum, whereas neither MASP-1 nor MASP-3 is required for alternative pathway function".

作者信息

Takahashi Minoru, Sekine Hideharu, Endo Yuichi, Fujita Teizo

出版信息

J Immunol. 2013 Mar 15;190(6):2477. doi: 10.4049/jimmunol.1390002.

DOI:10.4049/jimmunol.1390002
PMID:23456699
Abstract
摘要

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1
Comment on "Mannan-binding lectin-associated serine protease (MASP)-1 is crucial for lectin pathway activation in human serum, whereas neither MASP-1 nor MASP-3 is required for alternative pathway function".关于《甘露聚糖结合凝集素相关丝氨酸蛋白酶（MASP）-1对人血清中凝集素途径激活至关重要，而替代途径功能既不需要MASP-1也不需要MASP-3》的评论
J Immunol. 2013 Mar 15;190(6):2477. doi: 10.4049/jimmunol.1390002.
2
Response to comment on "Mannan-binding lectin-associated serine protease (MASP)-1 is crucial for lectin pathway activation in human serum, whereas neither MASP-1 nor MASP-3 is required for alternative pathway function".对“甘露聚糖结合凝集素相关丝氨酸蛋白酶（MASP）-1对人血清中凝集素途径的激活至关重要，而替代途径功能既不需要MASP-1也不需要MASP-3”一文评论的回应
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3
Comment on "the pro-factor D cleaving activity of MASP-1/-3 is not required for alternative pathway function".关于“旁路途径功能不需要MASP - 1/-3的前因子D裂解活性”的评论
J Immunol. 2014 Jun 15;192(12):5448-9. doi: 10.4049/jimmunol.1400766.
4
The pro-factor D cleaving activity of MASP-1/-3 is not required for alternative pathway function.旁路途径功能并不需要MASP-1/-3的前因子D裂解活性。
J Immunol. 2014 Jun 15;192(12):5447-8. doi: 10.4049/jimmunol.1400777.
5
Mannan-binding lectin-associated serine protease (MASP)-1 is crucial for lectin pathway activation in human serum, whereas neither MASP-1 nor MASP-3 is required for alternative pathway function.甘露聚糖结合凝集素相关丝氨酸蛋白酶（MASP）-1 对人血清中凝集素途径的激活至关重要，而 MASP-1 和 MASP-3 均不参与替代途径的功能。
J Immunol. 2012 Oct 15;189(8):3957-69. doi: 10.4049/jimmunol.1201736. Epub 2012 Sep 10.
6
The x-ray crystal structure of mannose-binding lectin-associated serine proteinase-3 reveals the structural basis for enzyme inactivity associated with the Carnevale, Mingarelli, Malpuech, and Michels (3MC) syndrome.甘露糖结合凝集素相关丝氨酸蛋白酶-3 的 X 射线晶体结构揭示了与卡内瓦莱、明加雷利、马尔普埃克和米歇尔（3MC）综合征相关酶无活性的结构基础。
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7
Essential Roles for Mannose-Binding Lectin-Associated Serine Protease-1/3 in the Development of Lupus-Like Glomerulonephritis in MRL/ Mice.甘露糖结合凝集素相关丝氨酸蛋白酶-1/3 在 MRL/lpr 小鼠狼疮样肾小球肾炎发展中的必需作用。
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8
Cutting Edge: A New Player in the Alternative Complement Pathway, MASP-1 Is Essential for LPS-Induced, but Not for Zymosan-Induced, Alternative Pathway Activation.前沿：替代补体途径的新成员，MASP-1 对于 LPS 诱导的但不是对于酵母聚糖诱导的替代途径激活是必需的。
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Cutting Edge: Role of MASP-3 in the Physiological Activation of Factor D of the Alternative Complement Pathway.前沿：甘露聚糖结合凝集素相关丝氨酸蛋白酶 3 在替代补体途径中因子 D 生理激活中的作用。
J Immunol. 2019 Sep 15;203(6):1411-1416. doi: 10.4049/jimmunol.1900605. Epub 2019 Aug 9.
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Novel mutation in MASP1 gene in a new family with 3MC syndrome.一个新的患有3MC综合征的家族中MASP1基因的新型突变。
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引用本文的文献

1
Key Components of the Complement Lectin Pathway Are Not Only Required for the Development of Inflammatory Arthritis but Also Regulate the Transcription of Factor D.补体凝集素途径的关键成分不仅是炎症性关节炎发展所必需的，而且还调节因子 D 的转录。
Front Immunol. 2020 Feb 21;11:201. doi: 10.3389/fimmu.2020.00201. eCollection 2020.
2
Complement in the Initiation and Evolution of Rheumatoid Arthritis.补体在类风湿关节炎的发生和演变中的作用。
Front Immunol. 2018 May 28;9:1057. doi: 10.3389/fimmu.2018.01057. eCollection 2018.
3
Essential role for the lectin pathway in collagen antibody-induced arthritis revealed through use of adenovirus programming complement inhibitor MAp44 expression.
通过使用腺病毒编程补体抑制剂MAp44表达揭示凝集素途径在胶原抗体诱导的关节炎中的重要作用。
J Immunol. 2014 Sep 1;193(5):2455-68. doi: 10.4049/jimmunol.1400752. Epub 2014 Jul 28.
4
C3 dysregulation due to factor H deficiency is mannan-binding lectin-associated serine proteases (MASP)-1 and MASP-3 independent in vivo.C3 调控异常是由于补体因子 H 缺乏，这在体内与甘露聚糖结合凝集素相关丝氨酸蛋白酶（MASP）-1 和 MASP-3 无关。
Clin Exp Immunol. 2014 Apr;176(1):84-92. doi: 10.1111/cei.12244.
5
Roles of adipocytes and fibroblasts in activation of the alternative pathway of complement in inflammatory arthritis in mice.脂肪细胞和成纤维细胞在小鼠炎症性关节炎补体替代途径激活中的作用。
J Immunol. 2013 Jun 15;190(12):6423-33. doi: 10.4049/jimmunol.1300580. Epub 2013 May 6.