Gene-Gene Interaction between LCE and CLEC16A Increases the Risk of Psoriasis in a Chinese Population.
作者信息
Wang Jianbo, Ding Yantao, Fan Xing, Zuo Xianbo, Wang Wen, Cheng Hui, Li Ping, Sun Liangdan, Yang Sen, Zhang Xuejun
机构信息
Institute of Dermatology, Department of Dermatology, No.1 Hospital, Anhui Medical University, Anhui, China.
Key Laboratory of Dermatology, Anhui Medical University, Ministry of Education, Hefei, Anhui, China.
出版信息
Ann Dermatol. 2014 Jun;26(3):421-3. doi: 10.5021/ad.2014.26.3.421. Epub 2014 Jun 12.
Abstract
摘要
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Int J Dermatol. 2012 Apr;51(4):389-95; quiz 395-8. doi: 10.1111/j.1365-4632.2011.05154.x.
2
Shared genetic factors may not explain the raised risk of comorbid inflammatory diseases in multiple sclerosis.共享的遗传因素可能无法解释多发性硬化症中并发炎症性疾病风险增加的原因。
Mult Scler. 2012 Oct;18(10):1430-6. doi: 10.1177/1352458512438240. Epub 2012 Mar 14.
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TALE homeodomain proteins regulate site-specific terminal differentiation, LCE genes and epidermal barrier.TALE 同源结构域蛋白调节特定部位的终末分化、LCE 基因和表皮屏障。
J Cell Sci. 2011 May 15;124(Pt 10):1681-90. doi: 10.1242/jcs.077552. Epub 2011 Apr 21.
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Am J Pathol. 2011 Apr;178(4):1470-7. doi: 10.1016/j.ajpath.2010.12.017.
5
Variants in MHC, LCE and IL12B have epistatic effects on psoriasis risk in Chinese population.MHC、LCE 和 IL12B 中的变异与中国人银屑病发病风险存在上位效应。
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6
Meta-analysis confirms the LCE3C_LCE3B deletion as a risk factor for psoriasis in several ethnic groups and finds interaction with HLA-Cw6.荟萃分析证实 LCE3C_LCE3B 缺失是多个族群罹患银屑病的风险因素，并发现与 HLA-Cw6 存在相互作用。
J Invest Dermatol. 2011 May;131(5):1105-9. doi: 10.1038/jid.2010.350. Epub 2010 Nov 25.
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