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Malaria and vascular endothelium.疟疾与血管内皮
Arq Bras Cardiol. 2014 Aug;103(2):165-9. doi: 10.5935/abc.20140088. Epub 2014 Jul 9.
2
Cytoadherence and virulence - the case of Plasmodium knowlesi malaria.细胞黏附和毒力——以疟原虫 knowlesi 疟疾为例。
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Inhibition of Infected Red Blood Cell Binding to the Vascular Endothelium.抑制感染的红细胞与血管内皮的结合。
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PECAM-1/CD31, an endothelial receptor for binding Plasmodium falciparum-infected erythrocytes.PECAM-1/CD31，一种用于结合恶性疟原虫感染红细胞的内皮受体。
Nat Med. 1997 Dec;3(12):1405-8. doi: 10.1038/nm1297-1405.
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Molecular basis of severe malaria.重症疟疾的分子基础
Proc Natl Acad Sci U S A. 2012 Jun 26;109(26):10130-1. doi: 10.1073/pnas.1207174109. Epub 2012 Jun 7.
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Plasmodium falciparum malaria--sticky jams and PECAM pie.恶性疟原虫疟疾——粘性堵塞与血小板内皮细胞黏附分子馅饼
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Ultrastructural aspects of Plasmodium falciparum-infected erythrocyte adherence to endothelial cells of Saimiri brain microvasculature.恶性疟原虫感染的红细胞与松鼠猴脑微血管内皮细胞黏附的超微结构特征
Am J Trop Med Hyg. 1996 Feb;54(2):169-77. doi: 10.4269/ajtmh.1996.54.169.
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In vivo sequestration of Plasmodium falciparum-infected human erythrocytes: a severe combined immunodeficiency mouse model for cerebral malaria.恶性疟原虫感染的人类红细胞的体内隔离：一种用于脑型疟疾的严重联合免疫缺陷小鼠模型。
J Exp Med. 1995 Sep 1;182(3):643-53. doi: 10.1084/jem.182.3.643.
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Plasmodium coatneyi-infected rhesus monkeys: a primate model for human cerebral malaria.感染柯氏疟原虫的恒河猴：一种人类脑型疟疾的灵长类动物模型。
Mem Inst Oswaldo Cruz. 1992;87 Suppl 3:443-7. doi: 10.1590/s0074-02761992000700074.

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Gymnema inodorum Leaf Extract Improves Cardiac Function in Experimental Mice Infected with Plasmodium Berghei.黄花倒水莲叶提取物可改善感染伯氏疟原虫的实验小鼠的心脏功能。
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Prevalence of Cardiovascular Complications in Malaria: A Systematic Review and Meta-Analysis.疟疾患者心血管并发症的患病率：系统评价和荟萃分析。
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Reduction in serum sphingosine 1-phosphate concentration in malaria.疟疾患者血清中1-磷酸鞘氨醇浓度降低。
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本文引用的文献

1
Persistently elevated laboratory markers of thrombosis and fibrinolysis after clinical recovery in malaria points to residual and smouldering cellular damage.疟疾临床康复后，血栓形成和纤维蛋白溶解的实验室指标持续升高，这表明存在残留且隐匿的细胞损伤。
Indian J Hematol Blood Transfus. 2012 Mar;28(1):29-36. doi: 10.1007/s12288-011-0106-y. Epub 2011 Sep 10.
2
Acute myocarditis in vivax malaria: an extremely rare complication.间日疟中的急性心肌炎：一种极其罕见的并发症。
Trop Doct. 2013 Jan;43(1):35-6. doi: 10.1177/0049475512473601. Epub 2013 Feb 26.
3
Duffy Blood Group System and the malaria adaptation process in humans.达菲血型系统与人类的疟疾适应过程
Rev Bras Hematol Hemoter. 2011;33(1):55-64. doi: 10.5581/1516-8484.20110016.
4
Malaria and the heart.疟疾与心脏
BMJ Case Rep. 2012 Nov 27;2012:bcr2012007275. doi: 10.1136/bcr-2012-007275.
5
Severity of Plasmodium vivax malaria in Karachi: a cross-sectional study.卡拉奇间日疟原虫疟疾的严重程度：一项横断面研究。
J Infect Dev Ctries. 2012 Sep 17;6(9):664-70. doi: 10.3855/jidc.2107.
6
Postmortem characterization of patients with clinical diagnosis of Plasmodium vivax malaria: to what extent does this parasite kill?临床诊断为间日疟原虫疟疾患者的死后特征：这种寄生虫的致死程度有多大？
Clin Infect Dis. 2012 Oct;55(8):e67-74. doi: 10.1093/cid/cis615. Epub 2012 Jul 6.
7
Functional analysis of Plasmodium vivax VIR proteins reveals different subcellular localizations and cytoadherence to the ICAM-1 endothelial receptor.对间日疟原虫 VIR 蛋白的功能分析揭示了不同的亚细胞定位和对细胞间黏附分子-1 内皮受体的细胞黏附作用。
Cell Microbiol. 2012 Mar;14(3):386-400. doi: 10.1111/j.1462-5822.2011.01726.x. Epub 2011 Dec 28.
8
On cytoadhesion of Plasmodium vivax: raison d'être?关于间日疟原虫的细胞黏附：存在的理由？
Mem Inst Oswaldo Cruz. 2011 Aug;106 Suppl 1:79-84. doi: 10.1590/s0074-02762011000900010.
9
Nitric oxide and the cardiovascular system: cell activation, vascular reactivity and genetic variant.一氧化氮与心血管系统：细胞激活、血管反应性与遗传变异。
Arq Bras Cardiol. 2011 Jan;96(1):68-75.
10
[Molecular basis of red blood cell adhesion to endothelium].[红细胞与内皮细胞黏附的分子基础]
Ann Pharm Fr. 2011 Jan;69(1):3-6. doi: 10.1016/j.pharma.2010.10.003. Epub 2010 Dec 3.

疟疾与血管内皮

Malaria and vascular endothelium.

作者信息

Alencar Filho Aristóteles Comte de, Lacerda Marcus Vinícius Guimarães de, Okoshi Katashi, Okoshi Marina Politi

机构信息

Universidade Federal do Amazonas, Manaus, AM, Brazil.

Fundação de Medicina Tropical Dr. Heitor Vieira Dourado, Manaus, AM, Brazil.

出版信息

Arq Bras Cardiol. 2014 Aug;103(2):165-9. doi: 10.5935/abc.20140088. Epub 2014 Jul 9.

DOI:10.5935/abc.20140088
PMID:25014058
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4150669/
Abstract

Involvement of the cardiovascular system in patients with infectious and parasitic diseases can result from both intrinsic mechanisms of the disease and drug intervention. Malaria is an example, considering that the endothelial injury by Plasmodium-infected erythrocytes can cause circulatory disorders. This is a literature review aimed at discussing the relationship between malaria and endothelial impairment, especially its effects on the cardiovascular system. We discuss the implications of endothelial aggression and the interdisciplinarity that should guide the malaria patient care, whose acute infection can contribute to precipitate or aggravate a preexisting heart disease.

摘要

感染性和寄生虫病患者的心血管系统受累可能源于疾病的内在机制和药物干预。疟疾就是一个例子，因为感染疟原虫的红细胞造成的内皮损伤可导致循环系统紊乱。这是一篇旨在探讨疟疾与内皮功能损害之间关系的文献综述，尤其是其对心血管系统的影响。我们讨论了内皮损伤的影响以及指导疟疾患者护理的跨学科问题，疟疾急性感染可能促使或加重已有的心脏病。