• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Evaluation of the diagnostic characteristics of urinary kidney injury molecule 1 (uKIM-1) and uKIM-1/creatinine ratio in the assessment of incipient diabetic kidney disease.

作者信息

De Carvalho José Antonio M, Tatsch Etiane, Hausen Bruna S, Bollick Yãnaí S, Torbitz Vanessa D, Guarda Naiara S, De Campos Luízi P, Duarte Thiago, Duarte Marta M M F, Londero Sílvia W K, Comim Fabio V, Moresco Rafael N

出版信息

Clin Chem Lab Med. 2015 Feb;53(2):e51-4. doi: 10.1515/cclm-2014-0655.

DOI:10.1515/cclm-2014-0655
PMID:25153602
Abstract
摘要

相似文献

1
Evaluation of the diagnostic characteristics of urinary kidney injury molecule 1 (uKIM-1) and uKIM-1/creatinine ratio in the assessment of incipient diabetic kidney disease.评估尿肾损伤分子1（uKIM-1）及uKIM-1/肌酐比值在早期糖尿病肾病评估中的诊断特征。
Clin Chem Lab Med. 2015 Feb;53(2):e51-4. doi: 10.1515/cclm-2014-0655.
2
Urinary kidney injury molecule-1 and neutrophil gelatinase-associated lipocalin as indicators of tubular damage in normoalbuminuric patients with type 2 diabetes.尿肾损伤分子-1和中性粒细胞明胶酶相关脂质运载蛋白作为2型糖尿病正常白蛋白尿患者肾小管损伤的指标
Clin Biochem. 2016 Feb;49(3):232-6. doi: 10.1016/j.clinbiochem.2015.10.016. Epub 2015 Oct 27.
3
Evaluation of the urinary kidney injury molecule-1 levels in patients with diabetic nephropathy.糖尿病肾病患者尿肾损伤分子-1水平的评估
Clin Invest Med. 2014 Dec 1;37(6):E377-83. doi: 10.25011/cim.v37i6.22242.
4
Urinary L-FABP and anaemia: distinct roles of urinary markers in type 2 diabetes.尿 L-FABP 与贫血：尿标志物在 2 型糖尿病中的不同作用。
Eur J Clin Invest. 2010 Feb;40(2):95-102. doi: 10.1111/j.1365-2362.2009.02220.x. Epub 2009 Nov 11.
5
Measuring urinary tubular biomarkers in type 2 diabetes does not add prognostic value beyond established risk factors.在 2 型糖尿病中检测尿肾小管生物标志物并不能提供比既定危险因素更多的预后价值。
Kidney Int. 2012 Oct;82(7):812-8. doi: 10.1038/ki.2012.218. Epub 2012 Jun 20.
6
Kidney injury molecule-1 as a promising biomarker for acute kidney injury in premature babies.肾损伤分子-1 作为早产儿急性肾损伤的有前途的生物标志物。
Am J Perinatol. 2013 Mar;30(3):245-52. doi: 10.1055/s-0032-1323587. Epub 2012 Aug 8.
7
Nonalbuminuric proteinuria as a biomarker for tubular damage in early development of nephropathy with type 2 diabetic patients.非白蛋白尿性蛋白尿作为 2 型糖尿病肾病早期小管损伤的生物标志物。
Diabetes Metab Res Rev. 2014 Nov;30(8):736-41. doi: 10.1002/dmrr.2546.
8
Clinical implication of urinary tubular markers in the early stage of nephropathy with type 2 diabetic patients.尿小管标记物在 2 型糖尿病肾病早期的临床意义。
Diabetes Res Clin Pract. 2012 Aug;97(2):251-7. doi: 10.1016/j.diabres.2012.02.019. Epub 2012 Mar 21.
9
Analysis of a urinary biomarker panel for obstructive nephropathy and clinical outcomes.用于梗阻性肾病和临床结局的尿液生物标志物组分析
PLoS One. 2014 Nov 17;9(11):e112865. doi: 10.1371/journal.pone.0112865. eCollection 2014.
10
Circulating kidney injury molecule-1 as a biomarker of renal parameters in diabetic kidney disease.循环肾损伤分子-1 作为糖尿病肾病肾参数的生物标志物。
J Diabetes Investig. 2020 Mar;11(2):435-440. doi: 10.1111/jdi.13139. Epub 2019 Sep 21.

引用本文的文献

1
KIM-1 as a biomarker of kidney tubular damage in normoalbuminuric patients with type 2 diabetes mellitus and insulin resistance.在正常白蛋白尿的2型糖尿病和胰岛素抵抗患者中，Kim-1作为肾小管损伤的生物标志物。
J Bras Nefrol. 2025 Jul-Sep;47(3):e20240123. doi: 10.1590/2175-8239-JBN-2024-0123en.
2
High Serum Uric Acid Is Associated with Tubular Damage and Kidney Inflammation in Patients with Type 2 Diabetes.血清尿酸水平升高与 2 型糖尿病患者的肾小管损伤和肾脏炎症有关。
Dis Markers. 2019 Apr 11;2019:6025804. doi: 10.1155/2019/6025804. eCollection 2019.
3
Urinary Biomarkers of Aminoglycoside-Induced Nephrotoxicity in Cystic Fibrosis: Kidney Injury Molecule-1 and Neutrophil Gelatinase-Associated Lipocalin.
囊性纤维化中氨基糖苷类诱导的肾毒性的尿生物标志物：肾损伤分子 1 和中性粒细胞明胶酶相关脂质运载蛋白。
Sci Rep. 2018 Mar 23;8(1):5094. doi: 10.1038/s41598-018-23466-4.