Ishige N, Laschi F, Pogliani L
Dipartimento di Chimica, Università della Calabria, Arcavacata di Rende (CS), Italy.
In Vivo. 1989 Jul-Aug;3(4):263-6.
In the course of a systematic in vivo P-31 NMR study of moderately insulted rat brains, some brains died after such injury. Changes in high-energy phosphate metabolism of these brains after injury were rationalised by the aid of a model based on the depletion of an insoluble Pi pool followed by depletion of PCr and ATP pools.
在对中度损伤大鼠脑进行的系统性体内磷-31核磁共振研究过程中,一些脑在这种损伤后死亡。借助一个基于不溶性无机磷池耗竭,随后磷酸肌酸和三磷酸腺苷池耗竭的模型,解释了这些脑损伤后高能磷酸代谢的变化。