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AH5183和西替地尔:两种抑制乙酰胆碱摄取到来自电鳐分离突触小泡的强效抑制剂。

AH5183 and cetiedil: two potent inhibitors of acetylcholine uptake into isolated synaptic vesicles from Torpedo marmorata.

作者信息

Diebler M F, Gaudry-Talarmain Y M

机构信息

Département de Neurochimie, CNRS, Gif-sur-Yvette, France.

出版信息

J Neurochem. 1989 Mar;52(3):813-21. doi: 10.1111/j.1471-4159.1989.tb02526.x.

Abstract

Synaptic vesicles purified on a sucrose-KCl sedimentation gradient were tested for their ability to accumulate [1-14C]acetylcholine ([1-14C]ACh) in the absence and in the presence of AH5183 and cetiedil. Kinetic studies of ACh transport showed that it was time dependent and saturable as a function of ACh concentration, with a KT of 1.2 mM. The protein-modifying agents N-ethylmaleimide and 4-chloro-7-nitrobenzo-2-oxa-1,3-diazole were powerful inhibitors of ACh uptake. In agreement with other studies, AH5183 was found to be a potent inhibitor of ACh uptake by synaptic vesicles. Inhibition was of the mixed noncompetitive type, and the inhibition constant was 45.2 +/- 3.4 nM. Cetiedil, a drug that resembles ACh, was previously shown on intact nerve endings to inhibit the translocation of newly synthesized ACh into the synaptic vesicle compartment, and we demonstrate here that cetiedil is indeed an efficient blocker of ACh uptake by isolated synaptic vesicles. It acted as a competitive inhibitor, with a Ki of 118.5 +/- 9.5 nM. Neither ATP-dependent calcium uptake nor Mg2+-ATPase activity was affected by the drugs, a finding showing their specificity toward the ACh uptake process. The binding of L-[3H]AH5183 to intact vesicles was characterized in the absence or the presence of ACh or cetiedil. Saturation experiments showed a total binding capacity of approximately 126 pmol/mg of vesicular protein and a dissociation constant of 19.9 +/- 4.1 nM under control conditions.(ABSTRACT TRUNCATED AT 250 WORDS)

摘要

在蔗糖 - 氯化钾沉降梯度上纯化的突触小泡,在不存在和存在AH5183及西替地尔的情况下,测试其积累[1 - 14C]乙酰胆碱([1 - 14C]ACh)的能力。ACh转运的动力学研究表明,其具有时间依赖性,并且作为ACh浓度的函数是可饱和的,转运常数(KT)为1.2 mM。蛋白质修饰剂N - 乙基马来酰亚胺和4 - 氯 - 7 - 硝基苯并 - 2 - 恶唑 - 1,3 - 二唑是ACh摄取的强力抑制剂。与其他研究一致,发现AH5183是突触小泡摄取ACh的有效抑制剂。抑制作用为混合非竞争性类型,抑制常数为45.2±3.4 nM。西替地尔是一种类似于ACh的药物,先前在完整神经末梢上显示可抑制新合成的ACh转运到突触小泡区室,我们在此证明西替地尔确实是分离的突触小泡摄取ACh的有效阻滞剂。它作为竞争性抑制剂,抑制常数(Ki)为118.5±9.5 nM。药物对ATP依赖性钙摄取和Mg2 + - ATP酶活性均无影响,这一发现表明它们对ACh摄取过程具有特异性。在不存在或存在ACh或西替地尔的情况下,对L - [3H]AH5183与完整小泡的结合进行了表征。饱和实验表明,在对照条件下,总结合能力约为126 pmol/mg小泡蛋白,解离常数为19.9±4.1 nM。(摘要截断于250字)

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