Yamasaki S, Amamoto Y, Imamura H, Tsuji Y
Acta Paediatr Jpn. 1989 Apr;31(2):198-204.
Kawasaki disease has recently been considered as a major cause of acquired valvular diseases in infants and children. But there have been few reports on the clinical course of the lesions caused by this disease, particularly aortic regurgitation. In February 1982, we examined a six-month-old male infant who had Sellers third degree aortic regurgitation (AR) and Sellers first degree mitral regurgitation (MR) without discernible dilatation, aneurysm formation or stenosis of the coronary arteries, and in January 1987 we performed a second cardiac catheterization in order to estimate the progress of these lesions. The results were: 1) His AR showed no apparent worsening; 2) His MR had completely recovered; 3) His volume and pressure studies were almost normal. The catheterization data were compatible with those of other clinical examinations. He no longer had cardiac enlargement as seen on a chest roentgenogram, and there were no signs of left ventricular hypertrophy on an electrocardiogram (ECG).
川崎病最近被认为是婴幼儿后天性瓣膜病的主要病因。但关于该疾病所致病变的临床病程，尤其是主动脉瓣反流的报道较少。1982年2月，我们检查了一名6个月大的男婴，他患有塞勒斯三级主动脉瓣反流（AR）和塞勒斯一级二尖瓣反流（MR），冠状动脉无明显扩张、动脉瘤形成或狭窄。1987年1月，我们进行了第二次心导管检查，以评估这些病变的进展情况。结果如下：1）他的主动脉瓣反流无明显恶化；2）他的二尖瓣反流已完全恢复；3）他的容量和压力研究基本正常。心导管检查数据与其他临床检查结果相符。他的胸部X线片上不再有心脏扩大的表现，心电图（ECG）上也没有左心室肥厚的迹象。
