Weinmann-Dorsch C, Koschel K
Institut für Virologie und Immunbiologie, Universität Würzburg, FRG.
FEBS Lett. 1989 Apr 24;247(2):185-8. doi: 10.1016/0014-5793(89)81330-4.
C6 rat glioma cells persistently infected with subacute sclerosing panencephalitis virus (C6/SSPE) were treated with measles antiserum and purified anti-measles IgG. This stimulated phosphoinositide breakdown and an increase in inositol phosphates. In uninfected C6 cells, however, only fetal calf serum (FCS), but not measles antiserum could induce inositol polyphosphate production.
用麻疹抗血清和纯化的抗麻疹IgG处理持续感染亚急性硬化性全脑炎病毒的C6大鼠胶质瘤细胞（C6/SSPE）。这刺激了磷酸肌醇分解并使肌醇磷酸增加。然而，在未感染的C6细胞中，只有胎牛血清（FCS）能诱导肌醇多磷酸的产生，而麻疹抗血清则不能。
