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Altering macrophage polarization in the tumor environment: the role of response gene to complement 32.

作者信息

Santoni Matteo, Cascinu Stefano, Mills Charles D

机构信息

Clinica di Oncologia Medica, AOU Ospedali Riuniti, Università Politecnica delle Marche, Ancona, Italy.

BioMedical Consultants, Marine on St. Croix, MN, USA.

出版信息

Cell Mol Immunol. 2015 Nov;12(6):783-4. doi: 10.1038/cmi.2014.115. Epub 2014 Nov 24.

DOI:10.1038/cmi.2014.115

PMID:25418471

原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC4716620/

Abstract

摘要

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本文引用的文献

Response gene to complement 32 (RGC-32) expression on M2-polarized and tumor-associated macrophages is M-CSF-dependent and enhanced by tumor-derived IL-4.

Cell Mol Immunol. 2015 Nov;12(6):692-9. doi: 10.1038/cmi.2014.108. Epub 2014 Nov 24.

M1 and M2 macrophages: the chicken and the egg of immunity.

J Innate Immun. 2014;6(6):716-26. doi: 10.1159/000364945. Epub 2014 Aug 13.

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CSF-1R inhibition alters macrophage polarization and blocks glioma progression.

Nat Med. 2013 Oct;19(10):1264-72. doi: 10.1038/nm.3337. Epub 2013 Sep 22.

M1 and M2 Macrophages: Oracles of Health and Disease.

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Altering macrophage polarization in the tumor environment: the role of response gene to complement 32.

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