Sutherlin Mark A, Hart Joseph M
Dept of Kinesiology, University of Virginia, Charlottesville, VA.
J Sport Rehabil. 2015 Feb;24(1):51-61. doi: 10.1123/jsr.2013-0112. Epub 2014 Dec 4.
Individuals with a history of low back pain (LBP) may present with decreased hip-abduction strength and increased trunk or gluteus maximus (GMax) fatigability. However, the effect of hip-abduction exercise on hip-muscle function has not been previously reported.
To compare hip-abduction torque and muscle activation of the hip, thigh, and trunk between individuals with and without a history of LBP during repeated bouts of side-lying hip-abduction exercise.
Repeated measures.
Clinical laboratory.
12 individuals with a history of LBP and 12 controls.
Repeated 30-s hip-abduction contractions.
Hip-abduction torque, normalized root-mean-squared (RMS) muscle activation, percent RMS muscle activation, and forward general linear regression.
Hip-abduction torque reduced in all participants as a result of exercise (1.57 ± 0.36 Nm/kg, 1.12 ± 0.36 Nm/kg; P < .001), but there were no group differences (F = 0.129, P = .723) or group-by-time interactions (F = 1.098, P = .358). All participants had increased GMax activation during the first bout of exercise (0.96 ± 1.00, 1.18 ± 1.03; P = .038). Individuals with a history of LBP had significantly greater GMax activation at multiple points during repeated exercise (P < .05) and a significantly lower percent of muscle activation for the GMax (P = .050) at the start of the third bout of exercise and for the biceps femoris (P = .039) at the end of exercise. The gluteal muscles best predicted hip-abduction torque in controls, while no consistent muscles were identified for individuals with a history of LBP.
Hip-abduction torque decreased in all individuals after hip-abduction exercise, although individuals with a history of LBP had increased GMax activation during exercise. Gluteal muscle activity explained hip-abduction torque in healthy individuals but not in those with a history of LBP. Alterations in hip-muscle function may exist in individuals with a history of LBP.
有腰痛（LBP）病史的个体可能存在髋外展力量下降以及躯干或臀大肌（GMax）疲劳性增加的情况。然而，此前尚未报道髋外展运动对髋部肌肉功能的影响。
比较有和没有LBP病史的个体在重复进行侧卧髋外展运动时的髋外展扭矩以及髋部、大腿和躯干的肌肉激活情况。
重复测量。
临床实验室。
12名有LBP病史的个体和12名对照组。
重复进行30秒的髋外展收缩。
髋外展扭矩、标准化均方根（RMS）肌肉激活、RMS肌肉激活百分比以及向前通用线性回归。
由于运动，所有参与者的髋外展扭矩均降低（1.57±0.36 Nm/kg，1.12±0.36 Nm/kg；P<.001），但不存在组间差异（F = 0.129，P = 0.723）或组×时间交互作用（F = 1.098，P = 0.358）。所有参与者在第一次运动时GMax激活均增加（0.96±1.00，1.18±1.03；P = 0.038）。有LBP病史的个体在重复运动期间多个时间点的GMax激活显著更高（P<.05），并且在第三次运动开始时GMax的肌肉激活百分比显著更低（P = 0.050），在运动结束时股二头肌的肌肉激活百分比显著更低（P = 0.039）。在对照组中，臀肌最能预测髋外展扭矩，而对于有LBP病史的个体，未发现一致的相关肌肉。
髋外展运动后所有个体的髋外展扭矩均降低，尽管有LBP病史的个体在运动期间GMax激活增加。臀肌活动可解释健康个体的髋外展扭矩，但不能解释有LBP病史个体的情况。有LBP病史的个体可能存在髋部肌肉功能改变。
