Łasecki Mateusz, Olchowy Cyprian, Pawluś Aleksander, Zaleska-Dorobisz Urszula
Department of Radiology, University of Medicine in Wrocław, Wrocław, Poland.
Pol J Radiol. 2014 Dec 14;79:467-71. doi: 10.12659/PJR.891393. eCollection 2014.
Ulnar neuropathy is the second most common peripheral nerve neuropathy after median neuropathy, with an incidence of 25 cases per 100 000 men and 19 cases per 100 000 women each year. Skipping (snapping) elbow syndrome is an uncommon cause of pain in the posterior-medial elbow area, sometimes complicated by injury of the ulnar nerve. One of the reason is the dislocation of the abnormal insertion of the medial triceps head over the medial epicondyle during flexion and extension movements. Others are: lack of the Osboune fascia leading to ulnar nerve instability and focal soft tissue tumors (fibromas, lipomas, etc). Recurrent subluxation of the nerve at the elbow results in a tractional and frictional neuritis with classical symptoms of peripheral neuralgia. As far as we know snapping triceps syndrome had never been evaluated in sonoelastography.
A 28yo semi-professional left handed tennis player was complaining about pain in posterior-medial elbow area. Initial US examination suggest golfers elbow syndrome which occurs quite commonly and has a prevalence of 0.3-0.6% in males and 0-3-1.1% in women and may be associated (approx. 50% of cases) with ulnar neuropathy. However subsequently made MRI revealed unusual distal triceps anatomy, moderate ulnar nerve swelling and lack of medial epicondylitis symptoms. Followed (second) US examination and sonoelastography have detected slipping of the both ulnar nerve and the additional band of the medial triceps head.
Snapping elbow syndrome is a poorly known medical condition, sometimes misdiagnosed as the medial epicondylitis. It describes a broad range of pathologies and anatomical abnormalities. One of the most often reasons is the slipping of the ulnar nerve as the result of the Osborne fascia/anconeus epitrochlearis muscle absence. Simultaneously presence of two or more "snapping reasons" is rare but should be always taken under consideration.
There are no sonoelastography studies describing golfers elbow syndrome, additional triceps band and ulnar neuritis. Our data suggest that the sonoelastography signs are similar to those seen in well described lateral epicondylitis syndrome, Achilles tendinitis and medial nerve neuralgia.
尺神经病变是仅次于正中神经病变的第二常见的周围神经病变,男性发病率为每年每10万人中有25例,女性为每年每10万人中有19例。跳跃(弹响)肘综合征是肘后内侧区域疼痛的罕见原因,有时会并发尺神经损伤。原因之一是在屈伸运动过程中,肱三头肌内侧头在内上髁的异常附着点脱位。其他原因包括:缺乏奥斯本筋膜导致尺神经不稳定以及局灶性软组织肿瘤(纤维瘤、脂肪瘤等)。神经在肘部反复半脱位会导致牵引性和摩擦性神经炎,并伴有周围神经痛的典型症状。据我们所知,弹响肱三头肌综合征从未在超声弹性成像中得到评估。
一名28岁的半职业左手网球运动员抱怨肘后内侧区域疼痛。最初的超声检查提示高尔夫球肘综合征,这种综合征相当常见,男性患病率为0.3 - 0.6%,女性为0.3 - 1.1%,并且可能与尺神经病变相关(约50%的病例)。然而,随后的磁共振成像显示肱三头肌远端解剖结构异常、尺神经中度肿胀且无内侧上髁炎症状。随后(第二次)超声检查和超声弹性成像检测到尺神经和肱三头肌内侧头的附加束带均有移位。
弹响肘综合征是一种鲜为人知的病症,有时会被误诊为内侧上髁炎。它描述了广泛的病理和解剖异常。最常见的原因之一是由于缺乏奥斯本筋膜/肱肌滑车肌导致尺神经移位。同时存在两个或更多“弹响原因”的情况很少见,但应始终予以考虑。
尚无关于高尔夫球肘综合征、肱三头肌附加束带和尺神经炎的超声弹性成像研究。我们的数据表明,超声弹性成像征象与在描述详尽的外侧上髁炎综合征、跟腱炎和正中神经神经痛中所见的征象相似。