Microvascular endothelial cells poised to take center stage in experimental renal fibrosis.
作者信息
Daehn Ilse, Bottinger Erwin P
机构信息
Charles Bronfman Institute for Personalized Medicine, and Division of Nephrology, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, New York
Charles Bronfman Institute for Personalized Medicine, and Division of Nephrology, Department of Medicine, Icahn School of Medicine at Mount Sinai, New York, New York.
出版信息
J Am Soc Nephrol. 2015 Apr;26(4):767-9. doi: 10.1681/ASN.2014121170. Epub 2014 Dec 22.
Abstract
摘要
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2
Endothelial NADPH oxidase-2 promotes interstitial cardiac fibrosis and diastolic dysfunction through proinflammatory effects and endothelial-mesenchymal transition.内皮型 NADPH 氧化酶-2 通过促炎作用和血管内皮-间质转化促进间质心脏纤维化和舒张功能障碍。
J Am Coll Cardiol. 2014 Jun 24;63(24):2734-41. doi: 10.1016/j.jacc.2014.02.572. Epub 2014 Mar 26.
3
Endothelial mitochondrial oxidative stress determines podocyte depletion in segmental glomerulosclerosis.内皮细胞线粒体氧化应激决定节段性肾小球硬化症中足细胞的耗竭。
J Clin Invest. 2014 Apr;124(4):1608-21. doi: 10.1172/JCI71195. Epub 2014 Mar 3.
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The endothelium as part of the integrative glomerular barrier complex.内皮细胞作为整合肾小球屏障复合体的一部分。
Kidney Int. 2014 Jan;85(1):8-11. doi: 10.1038/ki.2013.317.
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Mechanisms of hypoxia responses in renal tissue.肾脏组织缺氧反应的机制。
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