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Identification of key binding site residues of MCT1 for AR-C155858 reveals the molecular basis of its isoform selectivity.

作者信息

Nancolas B, Sessions R B, Halestrap A P

出版信息

Biochem J. 2015 Apr 1;467(1):192. doi: 10.1042/bj4670192.

DOI:10.1042/bj4670192
PMID:25793420
Abstract
摘要

相似文献

1
Identification of key binding site residues of MCT1 for AR-C155858 reveals the molecular basis of its isoform selectivity.鉴定MCT1与AR-C155858的关键结合位点残基揭示了其同工型选择性的分子基础。
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2
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Few Amino Acid Exchanges Expand the Substrate Spectrum of Monocarboxylate Transporter 10.少数氨基酸交换扩展了单羧酸转运蛋白10的底物谱。
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Mitochondrial Metabolism as a Treatment Target in Anaplastic Thyroid Cancer.
线粒体代谢作为间变性甲状腺癌的治疗靶点
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