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PR间期变异及非折返性室上性心动过速的异常机制,表现为经房室结双径路同时发生前向快径和慢径传导。

Unusual mechanism of PR interval variation and nonreentrant supraventricular tachycardia as manifestation of simultaneous anterograde fast and slow conduction through dual atrioventricular nodal pathways.

作者信息

Buss J, Kraatz J, Stegaru B, Neuss H, Heene D L

出版信息

Pacing Clin Electrophysiol. 1985 Mar;8(2):235-41. doi: 10.1111/j.1540-8159.1985.tb05755.x.

Abstract

Noninvasive recordings in a 69-year-old woman showed two distinct PR intervals of about 0.21 and 0.58 s, suggestive of dual AV nodal conduction. Various unusual mechanisms of transition from short to long and from long to short conduction intervals and phenomena of concealed conduction were due to the presence of two functionally separated intranodal pathways. Refractoriness of the slow pathway was associated with bradycardia. Episodes of tachycardia exhibited a one-to-two relationship between P-waves and ventricular activations as a consequence of simultaneous anterograde fast and slow conduction leading to double ventricular responses to single P-waves.

摘要

一名69岁女性的无创记录显示有两个不同的PR间期,分别约为0.21秒和0.58秒,提示房室结双径传导。从短传导间期到长传导间期以及从长传导间期到短传导间期的各种不寻常机制和隐匿性传导现象是由于存在两条功能上分离的结内通路。慢径路的不应期与心动过缓有关。心动过速发作时,由于快速径路和慢径路同时前传导致单个P波引起双心室反应,P波与心室激动之间呈现1:2关系。

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