血小板5-羟色胺能机制与肾小球肾炎
Platelet serotonergic mechanisms and glomerulonephritis.
作者信息
Mitra M K, Jain A K, Misra R, Sethi P P, Shukla R N, Chandra M, Verma M, Saxena A K, Shanker K
出版信息
Indian J Exp Biol. 1989 Jun;27(6):545-7.
Proven cases (28) of glomerulonephritis (GN) were evaluated for clinico-biochemical profile, bleeding time, platelet count, platelet aggregation and platelet serotonergic mechanisms. Inordinate increase in platelet aggregation and altered platelet serotonergic mechanisms (reduced 5-HT, uptake, reduced intraplatelet 5-HT and increased plasma 5-HT) were demonstrated in some histopathological types of GN. The mechanisms and importance of these alterations have been discussed.
对确诊的28例肾小球肾炎（GN）患者进行了临床生化指标、出血时间、血小板计数、血小板聚集及血小板5-羟色胺能机制评估。在某些组织病理学类型的GN中，发现血小板聚集过度增加且血小板5-羟色胺能机制改变（5-羟色胺摄取减少、血小板内5-羟色胺减少及血浆5-羟色胺增加）。已对这些改变的机制及重要性进行了讨论。
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