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房室结折返的手术治疗及解剖学引导手术的结果

Operative therapy of atrioventricular node reentry and results of an anatomically guided procedure.

作者信息

Fujimura O, Guiraudon G M, Yee R, Sharma A D, Klein G J

机构信息

Department of Medicine, University of Western Ontario, University Hospital, London, Canada.

出版信息

Am J Cardiol. 1989 Dec 1;64(19):1327-32. doi: 10.1016/0002-9149(89)90576-6.

Abstract

Operative therapy for atrioventricular (AV) node reentrant tachycardia consisting of dissection guided by anatomic landmarks is described. Of the 21 patients studied, 17 had the common type ("slow-fast") and 4 had the uncommon type ("fast-slow") of AV node reentry. Under normothermic cardio-pulmonary bypass, perinodal dissection was performed guided by anatomic landmarks: the atrial membranous septum, posterior superior process of the left ventricle, tendon of Todaro and os of the coronary sinus. There were no deaths or major complications. Seven to 10 days postoperatively, all patients had normal AV conduction except for one who continued to have AV node Wenckebach-type block. Postoperatively, the shortest cycle length capable of 1:1 conduction over the AV node changed from 323 +/- 66 to 421 +/- 90 ms (p less than 0.0001) anterogradely and from 330 +/- 86 to 449 +/- 164 ms (p = 0.004) retrogradely. Anterograde effective refractory period of the AV node prolonged from 264 +/- 49 to 358 +/- 107 ms (p = 0.012). Discontinuous AV conduction curves were no longer seen in 14 of 17 patients and 5 patients lost retrograde conduction. During follow-up (14.8 +/- 8.2 months), 19 patients have been free of arrhythmia without medication. Two patients required a second operation for recurrent tachycardia with success. No patient required a permanent pacemaker. These data show that operative therapy of AV node reentrant tachycardia can be guided by anatomic landmarks. Successful cure of tachycardia with perinodal dissection while preserving AV node conduction supports the view that the reentrant circuit is, at least in part, perinodal.

摘要

描述了一种基于解剖标志引导下的房室(AV)结折返性心动过速的手术治疗方法。在研究的21例患者中,17例为常见类型(“慢 - 快”型),4例为不常见类型(“快 - 慢”型)的房室结折返。在常温体外循环下,根据解剖标志进行结周解剖:房间隔膜部、左心室后上突、托达罗腱和冠状窦口。无死亡或严重并发症发生。术后7至10天,除1例仍有房室结文氏型阻滞外,所有患者房室传导正常。术后,房室结能够1:1传导的最短周期长度顺行从323±66毫秒变为421±90毫秒(p<0.0001),逆行从330±86毫秒变为449±164毫秒(p = 0.004)。房室结的顺行有效不应期从264±49毫秒延长至358±107毫秒(p = 0.012)。17例患者中有14例不再出现间断性房室传导曲线,5例患者失去逆行传导。在随访期间(14.8±8.2个月),19例患者无需药物治疗即无心律失常发作。2例患者因复发性心动过速需要二次手术,手术成功。无一例患者需要植入永久性起搏器。这些数据表明,房室结折返性心动过速的手术治疗可依据解剖标志进行。结周解剖成功治愈心动过速且保留房室结传导,支持了折返环路至少部分位于结周的观点。

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