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沉思、创伤后应激障碍和抑郁症状之间的关系。

The relationship between rumination, PTSD, and depression symptoms.

机构信息

University of Toledo, Department of Psychology, Toledo, OH, USA.

University of Toledo, Department of Psychology, Toledo, OH, USA; Department of Psychiatry & Human Behavior, Warren Alpert Medical School of Brown University, Providence, RI, USA.

出版信息

J Affect Disord. 2015 Jul 15;180:116-21. doi: 10.1016/j.jad.2015.04.006. Epub 2015 Apr 9.

Abstract

BACKGROUND

Posttraumatic stress disorder (PTSD) and major depressive disorder (MDD) are highly comorbid (Elhai et al., 2008. J. Clin. Psychiatry, 69, (4), 597-602). Rumination is a cognitive mechanism found to exacerbate and maintain both PTSD and MDD (Elwood et al., 2009. Clin. Psychol. Rev. 29, (1), 87-100; Olatunji et al., 2013. Clin. Psychol.: Sci. Pract. 20, (3), 225-257).

AIMS

Assess whether four rumination subtypes moderate the relationship between comorbid PTSD and MDD symptoms.

METHOD

We consecutively sampled patients (N=45) presenting to a mental health clinic using self-report measures of PTSD and MDD symptoms, and rumination in a cross-sectional design.

RESULTS

Repetitive rumination moderates the relationship between PTSD and MDD symptoms at one standard deviation above the mean (β=.044, p=.016), while anticipatory rumination moderates the relationship between PTSD and MDD symptoms at mean levels and higher levels of anticipatory rumination (mean β=.030, p=.042; higher β=.060, p=.008).

DISCUSSION

Repetitive and anticipatory rumination should be assessed in the context of comorbid PTSD and MDD and interventions should focus on reducing these rumination subtypes.

LIMITATIONS

Results should be replicated with other trauma populations because the number and complexity of traumatic events may impact the assessed symptoms. Constructs should also be assessed longitudinally, in order to establish causality. We are unable to confirm why rumination styles moderated the relationship between PTSD and depression or why counterfactual thinking and problem-focused thinking did not moderate the relationship between the two constructs.

摘要

背景

创伤后应激障碍(PTSD)和重度抑郁障碍(MDD)高度共病(Elhai 等人,2008 年。J. Clin. Psychiatry,69,(4),597-602)。反刍是一种认知机制,被发现会加剧和维持 PTSD 和 MDD(Elwood 等人,2009 年。Clin. Psychol. Rev. 29,(1),87-100;Olatunji 等人,2013 年。Clin. Psychol.: Sci. Pract. 20,(3),225-257)。

目的

评估四种反刍亚型是否调节 PTSD 和 MDD 症状共病的关系。

方法

我们使用 PTSD 和 MDD 症状的自我报告测量以及反刍的横断面设计,连续采样了到心理健康诊所就诊的患者(N=45)。

结果

重复反刍调节 PTSD 和 MDD 症状之间的关系,在平均值以上一个标准差处(β=0.044,p=0.016),而预期反刍调节 PTSD 和 MDD 症状之间的关系,在平均值和更高水平的预期反刍时(平均β=0.030,p=0.042;更高β=0.060,p=0.008)。

讨论

应在 PTSD 和 MDD 共病的背景下评估重复和预期反刍,并应侧重于减少这些反刍亚型。

局限性

由于创伤事件的数量和复杂性可能影响评估的症状,因此应该用其他创伤人群复制结果。为了建立因果关系,还应该对结构进行纵向评估。我们无法确定为什么反刍方式调节了 PTSD 和抑郁之间的关系,或者为什么反事实思维和问题聚焦思维没有调节这两个结构之间的关系。

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