Inhibition of hypertrophy is a good therapeutic strategy in ventricular pressure overload.
作者信息
Schiattarella Gabriele G, Hill Joseph A
机构信息
From Departments of Internal Medicine (Cardiology) (G.G.S., J.A.H.) and Molecular Biology (J.A.H.), University of Texas Southwestern Medical Center, Dallas, TX.
出版信息
Circulation. 2015 Apr 21;131(16):1435-47. doi: 10.1161/CIRCULATIONAHA.115.013894.
Abstract
摘要
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Inhibition of hypertrophy is a good therapeutic strategy in ventricular pressure overload.抑制心肌肥厚是治疗心室压力超负荷的一种有效策略。
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本文引用的文献
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Fibrosis--A Common Pathway to Organ Injury and Failure.纤维化——器官损伤与衰竭的共同途径。
N Engl J Med. 2015 Jul 2;373(1):96. doi: 10.1056/NEJMc1504848.
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Fibrosis--a common pathway to organ injury and failure.纤维化——器官损伤和衰竭的常见途径。
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Long noncoding RNAs and microRNAs in cardiovascular pathophysiology.长链非编码 RNA 和 microRNAs 在心血管病理生理学中的作用。
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Autophagy in cardiovascular biology.心血管生物学中的自噬
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Protein quality control and metabolism: bidirectional control in the heart.蛋白质质量控制与代谢：心脏中的双向调控
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Cardiovascular remodelling in coronary artery disease and heart failure.冠状动脉疾病和心力衰竭中的心血管重塑
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Cardiac macrophages and their role in ischaemic heart disease.心脏巨噬细胞及其在缺血性心脏病中的作用。
Cardiovasc Res. 2014 May 1;102(2):240-8. doi: 10.1093/cvr/cvu025. Epub 2014 Feb 5.
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Angiogenesis and cardiac hypertrophy: maintenance of cardiac function and causative roles in heart failure.血管生成与心肌肥厚：维持心功能及心力衰竭的致病作用。
Circ Res. 2014 Jan 31;114(3):565-71. doi: 10.1161/CIRCRESAHA.114.300507.
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Histone deacetylase inhibition blunts ischemia/reperfusion injury by inducing cardiomyocyte autophagy.组蛋白去乙酰化酶抑制通过诱导心肌细胞自噬减轻缺血/再灌注损伤。
Circulation. 2014 Mar 11;129(10):1139-51. doi: 10.1161/CIRCULATIONAHA.113.002416. Epub 2014 Jan 6.
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Pathological ventricular remodeling: therapies: part 2 of 2.病理性心室重塑：治疗方法：第2部分，共2部分
Circulation. 2013 Aug 27;128(9):1021-30. doi: 10.1161/CIRCULATIONAHA.113.001879.
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