Rogovoĭ Iu E, Gozhenko A I
Fiziol Zh (1978). 1989 Sep-Oct;35(5):18-23.
During the acute experimental nephritis a decrease of reabsorption by proximal tubules is combined with the activation of the lipid peroxidation into the renal cortex without changes in the activation of lysosomal enzyme of acid phosphatase and cathepsin D. Ionol, an inhibitor of oxygen products, exerts a protective action on the renal function and reabsorption by proximal tubules, decreasing concentration of the malondialdehyde into the renal cortex.
在急性实验性肾炎期间，近端肾小管重吸收的减少与肾皮质脂质过氧化的激活同时发生，而酸性磷酸酶和组织蛋白酶D等溶酶体酶的激活并无变化。氧产物抑制剂艾氏剂对肾功能和近端肾小管的重吸收具有保护作用，可降低肾皮质中丙二醛的浓度。
