Erol Kemal, Karahan Ali Yavuz, Kerimoğlu Ülkü, Ordahan Banu, Tekin Levent, Şahin Muhammed, Kaydok Ercan
Department of Physical Medicine and Rehabilitation, State Hospital , Nevsehir, Turkey.
Department of Physical Medicine and Rehabilitation, Beyhekim State Hospital , Konya, Turkey.
Clin Pract. 2015 Feb 5;5(1):699. doi: 10.4081/cp.2015.699. eCollection 2015 Jan 28.
Posterior tibial tendon dysfunction (PTTD) is an important cause of acquired pes planus that frequently observed in adults. Factors that play a role in the development of PTTD such as age-related tendon degeneration, inflammatory arthritis, hypertension, diabetes mellitus, obesity, peritendinous injections and more rarely acute traumatic rupture of the tendon. PTT is the primary dynamic stabilizer of medial arch of the foot. Plantar flexion and inversion of the foot occurs with contraction of tibialis posterior tendon, and arch of the foot becomes elaveted while midtarsal joints are locked and midfoot-hindfoot sets as rigid. Thus, during the walk gastrocnemius muscle works more efficiently. If the PTT does not work in the order, other foot ligaments and joint capsule would be increasingly weak and than pes planus occurs. We present a 10-year-old female patient diagnosed as PTTD and conservative treatment with review of the current literature.
胫后肌腱功能障碍（PTTD）是成人后天性平足症的一个重要原因。在PTTD发展过程中起作用的因素包括与年龄相关的肌腱退变、炎性关节炎、高血压、糖尿病、肥胖、腱周注射，以及较少见的肌腱急性创伤性断裂。胫后肌腱是足内侧纵弓的主要动态稳定器。当胫后肌腱收缩时，足跖屈和内翻，而中跗关节锁定且中足-后足固定为刚性时，足弓升高。因此，在行走过程中腓肠肌能更有效地工作。如果胫后肌腱不能正常发挥作用，其他足部韧带和关节囊会逐渐变弱，进而发生平足症。我们报告一名诊断为PTTD的10岁女性患者，并结合当前文献对保守治疗进行综述。
