Salimipour Hooman, Mehdizadeh Somayeh, Nemati Reza, Pourbehi Mohamad Reza, Pourbehi Gholam Reza, Assadi Majid
Department of Neurology, Bushehr University of Medical Sciences, Bushehr 7514633341, Iran.
Department of Neurology, Bushehr University of Medical Sciences, Bushehr 7514633341, Iran ; The Persian Gulf Tropical Medicine Research Center, Bushehr University of Medical Sciences, Bushehr 7514763448, Iran.
Case Rep Neurol Med. 2015;2015:924862. doi: 10.1155/2015/924862. Epub 2015 Apr 2.
Secondary erythrocytosis in cyanotic congenital heart disease (CCHD) is a compensatory response to chronic hypoxia which should be managed with caution. CCHD patients, who have compensated erythrocytosis but do not manifest significant neurologic symptoms, may experience secondary life-threatening complications such as stroke in case of inappropriate phlebotomy. This study reports a young man with CCHD who developed frequently repeated transient neurologic deficits with various presentations after one session of phlebotomy. The symptoms resolved a few days after the hematocrit (Hct) level returned to the prephlebotomy level.
青紫型先天性心脏病(CCHD)中的继发性红细胞增多症是对慢性缺氧的一种代偿性反应,对此应谨慎处理。患有代偿性红细胞增多症但未表现出明显神经症状的CCHD患者,若进行不适当的放血治疗,可能会出现如中风等危及生命的继发性并发症。本研究报告了一名患有CCHD的年轻男性,其在一次放血治疗后出现了频繁反复的各种短暂性神经功能缺损。这些症状在血细胞比容(Hct)水平恢复到放血前水平后的几天内消失。