Pearson Carolyn M, Wonderlich Stephen A, Smith Gregory T
Department of Psychiatry, University of Kentucky.
Department of Clinical Neuroscience, Neuropsychiatric Research Institute, University of North Dakota School of Medicine and Health Sciences.
Psychol Rev. 2015 Jul;122(3):516-35. doi: 10.1037/a0039268. Epub 2015 May 11.
This article offers a new model for bulimia nervosa (BN) that explains both the initial impulsive nature of binge eating and purging, as well as the compulsive quality of the fully developed disorder. The model is based on a review of advances in research on BN and advances in relevant basic psychological science. It integrates transdiagnostic personality risk, eating-disorder-specific risk, reinforcement theory, cognitive neuroscience, and theory drawn from the drug addiction literature. We identify both a state-based and a trait-based risk pathway, and we then propose possible state-by-trait interaction risk processes. The state-based pathway emphasizes depletion of self-control. The trait-based pathway emphasizes transactions between the trait of negative urgency (the tendency to act rashly when distressed) and high-risk psychosocial learning. We then describe a process by which initially impulsive BN behaviors become compulsive over time, and we consider the clinical implications of our model. (PsycINFO Database Record
本文提出了一种神经性贪食症(BN)的新模型,该模型既能解释暴饮暴食和催吐最初的冲动性质,也能解释完全发展型障碍的强迫性质。该模型基于对神经性贪食症研究进展及相关基础心理学进展的综述。它整合了跨诊断人格风险、饮食失调特异性风险、强化理论、认知神经科学以及来自药物成瘾文献的理论。我们识别出基于状态和基于特质的风险途径,然后提出可能的状态与特质相互作用风险过程。基于状态的途径强调自我控制的耗竭。基于特质的途径强调消极紧迫性特质(在痛苦时冲动行事的倾向)与高风险心理社会学习之间的相互作用。然后我们描述了一个过程,通过这个过程,最初冲动的神经性贪食症行为随着时间推移会变得强迫性,并且我们考虑了我们模型的临床意义。(PsycINFO数据库记录)