Micali N, De Stavola B, Ploubidis G, Simonoff E, Treasure J, Field A E
N. Micali, MD, PhD, University College London Institute of Child Health, Behavioural and Brain Sciences Unit, London; B. De Stavola, PhD, Centre for Statistical Methodology, London School of Hygiene and Tropical Medicine; G. Ploubidis, PhD, Department of Population Health, London School of Hygiene and Tropical Medicine, London; E. Simonoff, Department of Child and Adolescent Psychiatry, King's College London, Institute of Psychiatry and National Institute of Health Research Biomedical Research Unity for Mental Health, London; J. Treasure, MBBS, PhD, Eating Disorders Research Unit, Psychological Medicine, Institute of Psychiatry, King's College London, London, UK; A. E. Field, ScD, Division of Adolescent Medicine, Department of Medicine, Boston Children's Hospital and Harvard Medical School, Channing Division of Network Medicine, Brigham and Women's Hospital, and Department of Epidemiology, Harvard School of Public Health, Boston, Massachusetts, USA
N. Micali, MD, PhD, University College London Institute of Child Health, Behavioural and Brain Sciences Unit, London; B. De Stavola, PhD, Centre for Statistical Methodology, London School of Hygiene and Tropical Medicine; G. Ploubidis, PhD, Department of Population Health, London School of Hygiene and Tropical Medicine, London; E. Simonoff, Department of Child and Adolescent Psychiatry, King's College London, Institute of Psychiatry and National Institute of Health Research Biomedical Research Unity for Mental Health, London; J. Treasure, MBBS, PhD, Eating Disorders Research Unit, Psychological Medicine, Institute of Psychiatry, King's College London, London, UK; A. E. Field, ScD, Division of Adolescent Medicine, Department of Medicine, Boston Children's Hospital and Harvard Medical School, Channing Division of Network Medicine, Brigham and Women's Hospital, and Department of Epidemiology, Harvard School of Public Health, Boston, Massachusetts, USA.
Br J Psychiatry. 2015 Oct;207(4):320-7. doi: 10.1192/bjp.bp.114.152371. Epub 2015 Jul 23.
Eating disorder behaviours begin in adolescence. Few longitudinal studies have investigated childhood risk and protective FACTORS.
To investigate the prevalence of eating disorder behaviours and cognitions and associated childhood psychological, physical and parental risk factors among a cohort of 14-year-old children.
Data were collected from 6140 boys and girls aged 14 years. Gender-stratified models were used to estimate prospective associations between childhood body dissatisfaction, body mass index (BMI), self-esteem, maternal eating disorder and family economic disadvantage on adolescent eating disorder behaviours and cognitions.
Childhood body dissatisfaction strongly predicted eating disorder cognitions in girls, but only in interaction with BMI in boys. Higher self-esteem had a protective effect, particularly in boys. Maternal eating disorder predicted body dissatisfaction and weight/shape concern in adolescent girls and dieting in boys.
Risk factors for eating disorder behaviours and cognitions vary according to gender. Prevention strategies should be gender-specific and target modifiable predictors in childhood and early adolescence.
饮食失调行为始于青春期。很少有纵向研究调查儿童期的风险和保护因素。
调查一组14岁儿童中饮食失调行为和认知的患病率，以及相关的儿童期心理、身体和父母风险因素。
收集了6140名14岁男孩和女孩的数据。采用按性别分层的模型来估计儿童期身体不满意、体重指数（BMI）、自尊、母亲饮食失调和家庭经济劣势与青少年饮食失调行为和认知之间的前瞻性关联。
儿童期身体不满意强烈预测女孩的饮食失调认知，但在男孩中仅在与BMI的相互作用中起作用。较高的自尊有保护作用，尤其是在男孩中。母亲饮食失调预测青少年女孩的身体不满意和对体重/体型的关注，以及男孩的节食行为。
饮食失调行为和认知的风险因素因性别而异。预防策略应针对特定性别，并针对儿童期和青春期早期可改变的预测因素。
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