Heyman S N, Brezis M, Perlberg S, Metzner Y
Harefuah. 1989 Dec 1;117(11):358-60.
A 36-year-old woman who developed extreme erythrocytosis following partial correction of severe bilateral hydronephrosis by bilateral ureterolithotomy is presented. Hydronephrosis appears to stimulate renal synthesis and release of erythropoietin. The mechanism could involve increase in medullary hypoxia by increased pressure of the renal pelvis on the medullary vasculature. This might be especially marked when the renal blood supply is compromised by concomitant atherosclerosis, as in this case.
本文报告了一名36岁女性，她在双侧输尿管切开取石术部分纠正严重双侧肾积水后出现了极度红细胞增多症。肾积水似乎会刺激肾脏合成和释放促红细胞生成素。其机制可能包括肾盂对髓质血管系统压力增加导致髓质缺氧增加。当肾血供因合并动脉粥样硬化而受损时，这种情况可能会尤为明显，本病例即如此。
