Response to energy depletion: miR-451/AMPK loop.
作者信息
Bronisz Agnieszka, Chiocca E Antonio, Godlewski Jakub
机构信息
Department of Neurosurgery, Harvey Cushing Neuro-oncology Laboratories, Brigham and Women's Hospital, Harvard Medical School, Boston, MA, USA.
出版信息
Oncotarget. 2015 Jul 20;6(20):17851-2. doi: 10.18632/oncotarget.4606.
Abstract
摘要
相似文献
1
Response to energy depletion: miR-451/AMPK loop.对能量耗竭的反应：miR-451/AMPK 环路
Oncotarget. 2015 Jul 20;6(20):17851-2. doi: 10.18632/oncotarget.4606.
2
Glucose-based regulation of miR-451/AMPK signaling depends on the OCT1 transcription factor.基于葡萄糖的miR-451/AMPK信号调控依赖于OCT1转录因子。
Cell Rep. 2015 May 12;11(6):902-909. doi: 10.1016/j.celrep.2015.04.016. Epub 2015 Apr 30.
3
MicroRNA-451 regulates LKB1/AMPK signaling and allows adaptation to metabolic stress in glioma cells.microRNA-451 调节 LKB1/AMPK 信号通路并允许神经胶质瘤细胞适应代谢应激。
Mol Cell. 2010 Mar 12;37(5):620-32. doi: 10.1016/j.molcel.2010.02.018.
4
Strategies of eradicating glioma cells: a multi-scale mathematical model with MiR-451-AMPK-mTOR control.根除胶质瘤细胞的策略：一个具有MiR-451-AMPK-mTOR调控的多尺度数学模型
PLoS One. 2015 Jan 28;10(1):e0114370. doi: 10.1371/journal.pone.0114370. eCollection 2015.
5
AMPK: A metabolic checkpoint that regulates the growth of EGFR activated glioblastomas.腺苷酸活化蛋白激酶（AMPK）：一种调节表皮生长因子受体（EGFR）激活的胶质母细胞瘤生长的代谢检查点。
Cell Cycle. 2010 Jan 15;9(2):211-2. doi: 10.4161/cc.9.2.10540. Epub 2010 Jan 6.
6
The role of octamer binding transcription factors in glioblastoma multiforme.八聚体结合转录因子在多形性胶质母细胞瘤中的作用。
Biochim Biophys Acta. 2016 Jun;1859(6):805-11. doi: 10.1016/j.bbagrm.2016.03.003. Epub 2016 Mar 8.
7
CK2 inhibition induced PDK4-AMPK axis regulates metabolic adaptation and survival responses in glioma.CK2抑制诱导的PDK4-AMPK轴调节胶质瘤中的代谢适应和生存反应。
Exp Cell Res. 2016 May 15;344(1):132-142. doi: 10.1016/j.yexcr.2016.03.017. Epub 2016 Mar 18.
8
A computational multiscale model of glioblastoma growth: regulation of cell migration and proliferation via microRNA-451, LKB1 and AMPK.胶质母细胞瘤生长的计算多尺度模型：通过微小RNA-451、LKB1和AMPK对细胞迁移和增殖的调控
Annu Int Conf IEEE Eng Med Biol Soc. 2012;2012:6620-3. doi: 10.1109/EMBC.2012.6347512.
9
Strategies in regulating glioblastoma signaling pathways and anti-invasion therapy.调控胶质母细胞瘤信号通路和抗侵袭治疗的策略。
PLoS One. 2019 Apr 22;14(4):e0215547. doi: 10.1371/journal.pone.0215547. eCollection 2019.
10
AMP-activated protein kinase and cancer.腺苷酸活化蛋白激酶与癌症。
Acta Physiol (Oxf). 2009 May;196(1):55-63. doi: 10.1111/j.1748-1716.2009.01980.x. Epub 2009 Feb 25.
引用本文的文献
1
The nephrotoxin ochratoxin a impairs resilience of energy homeostasis of human proximal tubule cells.肾毒素赭曲霉毒素 A 损害人近端肾小管细胞能量稳态的弹性。
Mycotoxin Res. 2023 Nov;39(4):393-403. doi: 10.1007/s12550-023-00500-7. Epub 2023 Jul 19.
2
TFEB Signalling-Related MicroRNAs and Autophagy.TFEB 信号相关 microRNAs 和自噬。
Biomolecules. 2021 Jul 4;11(7):985. doi: 10.3390/biom11070985.
3
Hypoxic Roadmap of Glioblastoma-Learning about Directions and Distances in the Brain Tumor Environment.胶质母细胞瘤的缺氧路线图——了解脑肿瘤环境中的方向和距离
Cancers (Basel). 2020 May 13;12(5):1213. doi: 10.3390/cancers12051213.
4
Circulating RNAs as Potential Biomarkers in Amyotrophic Lateral Sclerosis.循环 RNA 作为肌萎缩侧索硬化症的潜在生物标志物。
Int J Mol Sci. 2020 Mar 3;21(5):1714. doi: 10.3390/ijms21051714.
5
MicroRNA-451 Inhibits Migration of Glioblastoma while Making It More Susceptible to Conventional Therapy.微小RNA-451抑制胶质母细胞瘤的迁移，同时使其对传统疗法更敏感。
Noncoding RNA. 2019 Mar 15;5(1):25. doi: 10.3390/ncrna5010025.
6
MicroRNA and extracellular vesicles in glioblastoma: small but powerful.胶质母细胞瘤中的微小RNA与细胞外囊泡：虽小却强大。
Brain Tumor Pathol. 2016 Apr;33(2):77-88. doi: 10.1007/s10014-016-0259-3. Epub 2016 Mar 11.
本文引用的文献
1
Glucose-based regulation of miR-451/AMPK signaling depends on the OCT1 transcription factor.基于葡萄糖的miR-451/AMPK信号调控依赖于OCT1转录因子。
Cell Rep. 2015 May 12;11(6):902-909. doi: 10.1016/j.celrep.2015.04.016. Epub 2015 Apr 30.
2
AMPK: positive and negative regulation, and its role in whole-body energy homeostasis.AMPK：正负调控及其在全身能量稳态中的作用。
Curr Opin Cell Biol. 2015 Apr;33:1-7. doi: 10.1016/j.ceb.2014.09.004. Epub 2014 Sep 26.
3
AMP-activated protein kinase (AMPK) beyond metabolism: a novel genomic stress sensor participating in the DNA damage response pathway.腺苷酸活化蛋白激酶（AMPK）超越代谢：一种参与DNA损伤反应途径的新型基因组应激传感器。
Cancer Biol Ther. 2014 Feb;15(2):156-69. doi: 10.4161/cbt.26726. Epub 2013 Nov 1.
4
Brain tumor initiating cells adapt to restricted nutrition through preferential glucose uptake.脑肿瘤起始细胞通过优先摄取葡萄糖来适应有限的营养供应。
Nat Neurosci. 2013 Oct;16(10):1373-82. doi: 10.1038/nn.3510. Epub 2013 Sep 1.
5
MicroRNA-451 regulates LKB1/AMPK signaling and allows adaptation to metabolic stress in glioma cells.microRNA-451 调节 LKB1/AMPK 信号通路并允许神经胶质瘤细胞适应代谢应激。
Mol Cell. 2010 Mar 12;37(5):620-32. doi: 10.1016/j.molcel.2010.02.018.
6
Stem cells, stress, metabolism and cancer: a drama in two Octs.干细胞、压力、新陈代谢与癌症：一部由两个Oct上演的“戏剧”
Trends Biochem Sci. 2009 Oct;34(10):491-9. doi: 10.1016/j.tibs.2009.06.003. Epub 2009 Sep 4.
7
Oct1 loss of function induces a coordinate metabolic shift that opposes tumorigenicity.Oct1功能丧失会引发一种与致瘤性相反的协调性代谢转变。
Nat Cell Biol. 2009 Mar;11(3):320-7. doi: 10.1038/ncb1840. Epub 2009 Feb 15.
Zotero 插件