Renvert Stefan, Quirynen Marc
Department of Oral Health Sciences, Kristianstad University, Kristianstad, Sweden.
School of Dental Sciences, Trinity College, Dublin, Ireland.
Clin Oral Implants Res. 2015 Sep;26 Suppl 11:15-44. doi: 10.1111/clr.12636.
To examine the existing evidence in identifying risk indicators in the etiology of peri-implantitis.
A literature search was performed in MEDLINE via PubMed database of the US National Library of Medicine, for articles published until October 2014 using Medical Subject Heading search terms + free text terms and in different combinations.
The microbiota associated with peri-implantitis is complex, demonstrating differences and similarities to the one seen at periodontitis sites. Plaque accumulation at dental implants triggers the inflammatory response leading to peri-implant mucositis/peri-implantitis. Individuals with a history of periodontal disease and smokers have an increased risk of developing peri-implantitis. There is some evidence to support the role of genetic polymorphism, diabetes, and excess cement as risk indicators for the development of peri-implantitis. There is also evidence to support that individuals on regular maintenance are less likely to develop peri-implantitis and that successful treatment of periodontitis prior to implant placement lowers the risk of peri-implantitis.
Plaque accumulation at implants will result in the development of an inflammation at implants. A history of periodontal disease, smoking, excess cement, and lack of supportive therapy should be considered as risk indicators for the development of peri-implantitis.
研究在种植体周围炎病因中识别风险指标的现有证据。
通过美国国立医学图书馆的PubMed数据库在MEDLINE中进行文献检索,检索截至2014年10月发表的文章,使用医学主题词检索词+自由文本词并采用不同组合。
与种植体周围炎相关的微生物群很复杂,与牙周炎部位所见的微生物群既有差异又有相似之处。牙种植体上的菌斑积聚引发炎症反应,导致种植体周围黏膜炎/种植体周围炎。有牙周病史的个体和吸烟者发生种植体周围炎的风险增加。有一些证据支持基因多态性、糖尿病和多余黏固剂作为种植体周围炎发生的风险指标的作用。也有证据支持定期维护的个体发生种植体周围炎的可能性较小,并且在种植体植入前成功治疗牙周炎可降低种植体周围炎的风险。
种植体上的菌斑积聚将导致种植体发生炎症。牙周病史、吸烟、多余黏固剂和缺乏支持性治疗应被视为种植体周围炎发生的风险指标。