[Interaction of insulin with lymphocyte plasma membrane receptors after experimental burn injury].

It was demonstrated in experiments on rats who has suffered from IIIa-IIIb degree thermal burns of the skin that changes in the function of the insular apparatus are accompanied by transitory diabetes caused by inhibition of insulin secretion and disturbance in insulin-receptor interrelations. In the period of stress the burn injury (in the first hours after the burn) leads to inhibition of insulin production in the animals, while hyperglycemia occurring in the later posttraumatic period results from inhibition of the insulin receptor interrelations and, evidently, from reduction of the postreceptor insulin action.