勘误：NLRP10是一种由树突状细胞启动适应性免疫所必需的NOD样受体。 - Suppr

Corrigendum: NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cells.

作者信息

Eisenbarth Stephanie C, Williams Adam, Colegio Oscar R, Meng Hailong, Strowig Till, Rongvaux Anthony, Henao-Mejia Jorge, Thaiss Christoph A, Joly Sophie, Gonzalez David G, Xu Lan, Zenewicz Lauren A, Haberman Ann M, Elinav Eran, Kleinstein Steven H, Sutterwala Fayyaz S, Flavell Richard A

出版信息

Nature. 2016 Feb 25;530(7591):504. doi: 10.1038/nature16074. Epub 2015 Nov 25.

DOI:10.1038/nature16074

PMID:26605525

Abstract

摘要

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Nature. 2012 Apr 25;484(7395):510-3. doi: 10.1038/nature11012.

DOCK8 is a Cdc42 activator critical for interstitial dendritic cell migration during immune responses.

Blood. 2012 May 10;119(19):4451-61. doi: 10.1182/blood-2012-01-407098. Epub 2012 Mar 28.

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Proc Natl Acad Sci U S A. 2012 Apr 10;109(15):E898-904. doi: 10.1073/pnas.1118155109. Epub 2012 Mar 19.

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Corrigendum: NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cells.

作者信息

出版信息

Nature. 2016 Feb 25;530(7591):504. doi: 10.1038/nature16074. Epub 2015 Nov 25.

DOI:10.1038/nature16074

PMID:26605525

Abstract

摘要

相似文献

Corrigendum: NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cells.

Nature. 2016 Feb 25;530(7591):504. doi: 10.1038/nature16074. Epub 2015 Nov 25.

Coincidental loss of DOCK8 function in NLRP10-deficient and C3H/HeJ mice results in defective dendritic cell migration.

Proc Natl Acad Sci U S A. 2015 Mar 10;112(10):3056-61. doi: 10.1073/pnas.1501554112. Epub 2015 Feb 23.

Innate Response to Human Cancer Cells with or without IL-2 Receptor Common γ-Chain Function in NOD Background Mice Lacking Adaptive Immunity.

J Immunol. 2015 Aug 15;195(4):1883-90. doi: 10.4049/jimmunol.1402103. Epub 2015 Jul 13.

Mechanisms and pathways of innate immune activation and regulation in health and cancer.

Hum Vaccin Immunother. 2014;10(11):3270-85. doi: 10.4161/21645515.2014.979640.

Roles of NLRP10 in innate and adaptive immunity.

Microbes Infect. 2013 Jun;15(6-7):516-23. doi: 10.1016/j.micinf.2013.03.008. Epub 2013 Apr 4.

NLRP10 enhances Shigella-induced pro-inflammatory responses.

Cell Microbiol. 2012 Oct;14(10):1568-83. doi: 10.1111/j.1462-5822.2012.01822.x. Epub 2012 Jun 21.

Indirect Toll-like receptor 5-mediated activation of conventional dendritic cells promotes the mucosal adjuvant activity of flagellin in the respiratory tract.

Vaccine. 2015 Jun 26;33(29):3331-41. doi: 10.1016/j.vaccine.2015.05.022. Epub 2015 May 21.

Dendritic cells as sensors of environmental perturbations.

Microbes Infect. 2008 Jul;10(9):990-4. doi: 10.1016/j.micinf.2008.07.013. Epub 2008 Jul 10.

Essential role of Rip2 in the modulation of innate and adaptive immunity triggered by Nod1 and Nod2 ligands.

Eur J Immunol. 2011 May;41(5):1445-55. doi: 10.1002/eji.201040827. Epub 2011 Apr 12.

The impact of eicosanoids on the crosstalk between innate and adaptive immunity: the key roles of dendritic cells.

Tissue Antigens. 2005 Jun;65(6):507-14. doi: 10.1111/j.1399-0039.2005.00394.x.

引用本文的文献

Epithelial Nlrp10 inflammasome mediates protection against intestinal autoinflammation.

Nat Immunol. 2023 Apr;24(4):585-594. doi: 10.1038/s41590-023-01450-z. Epub 2023 Mar 20.

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Sci Immunol. 2018 Feb 23;3(20). doi: 10.1126/sciimmunol.aam9841.

Mutations in fetal genes involved in innate immunity and host defense against microbes increase risk of preterm premature rupture of membranes (PPROM).

Mol Genet Genomic Med. 2017 Nov;5(6):720-729. doi: 10.1002/mgg3.330. Epub 2017 Aug 23.

NLRP10 Enhances CD4 T-Cell-Mediated IFNγ Response Regulation of Dendritic Cell-Derived IL-12 Release.

Front Immunol. 2017 Nov 2;8:1462. doi: 10.3389/fimmu.2017.01462. eCollection 2017.

Foundations of Immunometabolism and Implications for Metabolic Health and Disease.

Immunity. 2017 Sep 19;47(3):406-420. doi: 10.1016/j.immuni.2017.08.009.

本文引用的文献

Coincidental loss of DOCK8 function in NLRP10-deficient and C3H/HeJ mice results in defective dendritic cell migration.

Proc Natl Acad Sci U S A. 2015 Mar 10;112(10):3056-61. doi: 10.1073/pnas.1501554112. Epub 2015 Feb 23.

Addendum: defective Dock2 expression in a subset of ASC-deficient mouse lines.

Nat Immunol. 2012 Jul;13(7):701-2. doi: 10.1038/ni.2389.

NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cells.

Nature. 2012 Apr 25;484(7395):510-3. doi: 10.1038/nature11012.

DOCK8 is a Cdc42 activator critical for interstitial dendritic cell migration during immune responses.

Blood. 2012 May 10;119(19):4451-61. doi: 10.1182/blood-2012-01-407098. Epub 2012 Mar 28.

Spontaneous mutation of the Dock2 gene in Irf5-/- mice complicates interpretation of type I interferon production and antibody responses.

Proc Natl Acad Sci U S A. 2012 Apr 10;109(15):E898-904. doi: 10.1073/pnas.1118155109. Epub 2012 Mar 19.

The inflammasome adaptor ASC regulates the function of adaptive immune cells by controlling Dock2-mediated Rac activation and actin polymerization.

Nat Immunol. 2011 Sep 4;12(10):1010-6. doi: 10.1038/ni.2095.

Combined immunodeficiency associated with DOCK8 mutations.

N Engl J Med. 2009 Nov 19;361(21):2046-55. doi: 10.1056/NEJMoa0905506. Epub 2009 Sep 23.

Know thy mouse.

Trends Genet. 2006 Dec;22(12):649-53. doi: 10.1016/j.tig.2006.09.010. Epub 2006 Sep 26.

Corrigendum: NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cells.

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Corrigendum: NLRP10 is a NOD-like receptor essential to initiate adaptive immunity by dendritic cells.

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