Tanrivermis Sayit Asli, Aslan Kerim, Elmali Muzaffer, Gungor Inci
a Department of Radiology , Ondokuz Mayis University Faculty of Medicine , Samsun , Turkey and.
b Department of Ophthalmology , Ondokuz Mayis University Faculty of Medicine , Samsun , Turkey.
Cutan Ocul Toxicol. 2016 Dec;35(4):337-40. doi: 10.3109/15569527.2015.1122031. Epub 2016 Jan 28.
We report a 52-year-old man with methanol intoxication who showed optic nerve damage as assessed by magnetic resonance imaging (MRI). He was admitted to the hospital with blurred vision after the consumption of alcohol (600-700 ml of cologne). He was treated with intravenous ethanol, NaHCO3 and hemodialysis. On admission, a brain and orbital MRI was performed. Bilateral mild contrast enhancement was detected on the contrast-enhanced images in the retrobulbar segment of the optic nerves (RBONs). Also, diffusion-weighted images showed restricted diffusion in the RBONs. Diagnosis was considered as methanol-induced optic neuropathy based on the MRI findings of the optic nerves.
我们报告一名52岁甲醇中毒男性，磁共振成像（MRI）评估显示其存在视神经损伤。他在饮用酒精（600 - 700毫升古龙水）后因视力模糊入院。给予其静脉注射乙醇、碳酸氢钠并进行血液透析治疗。入院时，进行了脑部和眼眶MRI检查。在视神经球后段（RBONs）的增强图像上检测到双侧轻度对比增强。此外，扩散加权图像显示RBONs存在扩散受限。根据视神经的MRI表现，诊断为甲醇所致视神经病变。
