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Hyaluronidase 2 deficiency is a molecular cause of cor triatriatum sinister in mice.

作者信息

Chowdhury Biswajit, Xiang Bo, Muggenthaler Martina, Dolinsky Vernon W, Triggs-Raine Barbara

机构信息

Department of Biochemistry & Medical Genetics, University of Manitoba, 745 Bannatyne Ave., Winnipeg, MB R3E 0J9, Canada.

Pharmacology & Therapeutics, University of Manitoba, 745 Bannatyne Ave., Winnipeg, MB R3E 0J9, Canada; The Children's Hospital Research Institute of Manitoba, 715 McDermot Ave., Winnipeg, MB R3E 3P4.

出版信息

Int J Cardiol. 2016 Apr 15;209:281-3. doi: 10.1016/j.ijcard.2016.02.072. Epub 2016 Feb 10.

DOI:10.1016/j.ijcard.2016.02.072
PMID:26901790
Abstract
摘要

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Cor triatriatum sinister: a case series.左心房三房心：病例系列
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Anti-Inflammatory Effects of the 35kDa Hyaluronic Acid Fragment (B-HA/HA35).35kDa透明质酸片段（B-透明质酸/HA35）的抗炎作用
J Inflamm Res. 2023 Jan 13;16:209-224. doi: 10.2147/JIR.S393495. eCollection 2023.
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Cor Triatriatum Sinister Presenting as Cardioembolic Stroke in a Young Woman.一名年轻女性以心脏栓塞性中风为表现的左三房心
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Elucidating the clinical spectrum and molecular basis of HYAL2 deficiency.阐明 HYAL2 缺乏症的临床谱和分子基础。
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Hyaluronan: Metabolism and Function.透明质酸：代谢与功能。
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Hyaluronidase 2 Deficiency Causes Increased Mesenchymal Cells, Congenital Heart Defects, and Heart Failure.透明质酸酶2缺乏导致间充质细胞增多、先天性心脏缺陷和心力衰竭。
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