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KIF4A and PP2A-B56 form a spatially restricted feedback loop opposing Aurora B at the anaphase central spindle.
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2
PP2A-B56 opposes Mps1 phosphorylation of Knl1 and thereby promotes spindle assembly checkpoint silencing.
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3
Increased Aurora B activity causes continuous disruption of kinetochore-microtubule attachments and spindle instability.
Proc Natl Acad Sci U S A. 2014 Sep 23;111(38):E3996-4005. doi: 10.1073/pnas.1408017111. Epub 2014 Sep 8.
4
Depletion of IK causes mitotic arrest through aberrant regulation of mitotic kinases and phosphatases.
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5
Nuclear protein IK undergoes dynamic subcellular translocation and forms unique nuclear bodies during the cell cycle.
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Characterization of IK cytokine expression in mouse endometrium during early pregnancy and its significance on implantation.
Int J Mol Med. 2012 Sep;30(3):615-21. doi: 10.3892/ijmm.2012.1019. Epub 2012 Jun 11.
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RED, a spindle pole-associated protein, is required for kinetochore localization of MAD1, mitotic progression, and activation of the spindle assembly checkpoint.
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Activation of Aurora-A kinase by protein partner binding and phosphorylation are independent and synergistic.
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Formation of stable attachments between kinetochores and microtubules depends on the B56-PP2A phosphatase.
Nat Cell Biol. 2011 Aug 28;13(10):1265-71. doi: 10.1038/ncb2327.
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