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体内分流测试:脑室导管问题的观察性研究

Shunt Testing In Vivo: Observational Study of Problems with Ventricular Catheter.

作者信息

Czosnyka Zofia H, Sinha Rohitiwa, Morgan James A D, Wawrzynski James R, Price Steven J, Garnett Matthew, Pickard John D, Czosnyka M

机构信息

Division of Neurosurgery, Department of Clinical Neurosciences, University of Cambridge, Cambridge, UK.

出版信息

Acta Neurochir Suppl. 2016;122:353-6. doi: 10.1007/978-3-319-22533-3_69.

Abstract

Most shunt obstructions happen at the inlet of the ventricular catheter. Three hundred six infusion studies from 2007 to 2011 were classified as having a typical pattern of either proximal occlusion or patency. We describe different patterns of shunt ventricular obstruction.Solid block: Cerebrospinal fluid (CSF) aspiration was impossible. Baseline pressure was without pulse waveform (respiratory waveform may be visible). A quick increase of pressure to a level compatible with the shunt's setting was recorded in response to infusion. Distal occlusion of the shunt via transcutaneous compression resulted in a rapid increase in pressure to levels above 50 mmHg. This pattern was attributed to a solid ventricular block.Slit ventricles: At baseline, a pattern similar to that of the solid block was observed. After compression, the pressure increases, the pulse waveform appears, and the intracranial pressure is often stabilized at 25-40 mmHg. It is probable that previously slit ventricles were opened during the test.Partial block: In a partial block of the ventricular catheter by an in-growing choroid plexus, the pulse waveform at baseline was observed and CSF aspiration was possible. During infusion, the pressure increased, but the pulse amplitude disappeared. During the increase in the pressure in the shunt prechamber, the connection with the ventricles is disturbed by repositioning of the plexus.Infusion study via the shunt prechamber is able to visualize ventricular obstruction of the hydrocephalus shunt.

摘要

大多数分流梗阻发生在脑室导管的入口处。对2007年至2011年的306项灌注研究进行分类,结果显示具有近端阻塞或通畅的典型模式。我们描述了分流性脑室梗阻的不同模式。

实性阻塞

无法抽吸脑脊液(CSF)。基线压力无脉搏波形(呼吸波形可能可见)。灌注时记录到压力迅速升高至与分流装置设置相符的水平。经皮压迫导致分流远端阻塞时,压力迅速升高至50 mmHg以上。这种模式归因于实性脑室阻塞。

裂隙脑室

基线时,观察到与实性阻塞相似的模式。压迫后,压力升高,脉搏波形出现,颅内压常稳定在25 - 40 mmHg。很可能在测试过程中先前的裂隙脑室被打开。

部分阻塞

在脉络丛向内生长导致脑室导管部分阻塞时,观察到基线时的脉搏波形,并且可以抽吸脑脊液。灌注期间,压力升高,但脉搏幅度消失。在分流前房压力升高期间,脉络丛重新定位会干扰与脑室的连接。

通过分流前房进行灌注研究能够观察到脑积水分流的脑室梗阻情况。

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