Viglione G, Rivetti R, Lavagna F, Cartia Q
Minerva Chir. 1989 Mar 15;44(5):919-22.
A case of emergency revascularisation of the hepatic artery for liver necrosis following its forced ligature during corpo-caudal pancreatectomy for cancer of the pancreas in referred. Necrosis protection mechanisms, namely higher portal flow, increase in O2 extraction and collateral circulation are analysed. The therapeutic aids for preventing this serious event are presented. It is considered that peripheral expansion of the hepatic artery thrombosis prevented the installation of a valid collateral circulation, although the hypothesis of a reduction in portal flow should not be ignored.
本文报告了一例在胰体尾癌胰体尾切除术中肝动脉被迫结扎后，因肝坏死而行肝动脉紧急血运重建的病例。分析了坏死保护机制，即较高的门静脉血流、氧摄取增加和侧支循环。介绍了预防这一严重事件的治疗辅助措施。认为肝动脉血栓形成的外周扩展阻止了有效的侧支循环的建立，尽管门静脉血流减少的假设也不应被忽视。
