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前蛋白转化酶枯草杆菌蛋白酶/克新蛋白酶9型(PCSK9)单域抗体是低密度脂蛋白受体降解的强效抑制剂。

Proprotein Convertase Subtilisin/Kexin Type 9 (PCSK9) Single Domain Antibodies Are Potent Inhibitors of Low Density Lipoprotein Receptor Degradation.

作者信息

Weider Elodie, Susan-Resiga Delia, Essalmani Rachid, Hamelin Josée, Asselin Marie-Claude, Nimesh Surendra, Ashraf Yahya, Wycoff Keith L, Zhang Jianbing, Prat Annik, Seidah Nabil G

机构信息

From the Laboratory of Biochemical Neuroendocrinology, Institut de Recherches Cliniques de Montréal, University of Montreal, Montreal, Quebec H2W 1R7, Canada.

Planet Biotechnology Inc., Hayward, California 94545-2740, and.

出版信息

J Biol Chem. 2016 Aug 5;291(32):16659-71. doi: 10.1074/jbc.M116.717736. Epub 2016 Jun 8.

Abstract

Single domain antibodies (sdAbs) correspond to the antigen-binding domains of camelid antibodies. They have the same antigen-binding properties and specificity as monoclonal antibodies (mAbs) but are easier and cheaper to produce. We report here the development of sdAbs targeting human PCSK9 (proprotein convertase subtilisin/kexin type 9) as an alternative to anti-PCSK9 mAbs. After immunizing a llama with human PCSK9, we selected four sdAbs that bind PCSK9 with a high affinity and produced them as fusion proteins with a mouse Fc. All four sdAb-Fcs recognize the C-terminal Cys-His-rich domain of PCSK9. We performed multiple cellular assays and demonstrated that the selected sdAbs efficiently blocked PCSK9-mediated low density lipoprotein receptor (LDLR) degradation in cell lines, in human hepatocytes, and in mouse primary hepatocytes. We further showed that the sdAb-Fcs do not affect binding of PCSK9 to the LDLR but rather block its induced cellular LDLR degradation. Pcsk9 knock-out mice expressing a human bacterial artificial chromosome (BAC) transgene were generated, resulting in plasma levels of ∼300 ng/ml human PCSK9. Mice were singly or doubly injected with the best sdAb-Fc and analyzed at day 4 or 11, respectively. After 4 days, mice exhibited a 32 and 44% decrease in the levels of total cholesterol and apolipoprotein B and ∼1.8-fold higher liver LDLR protein levels. At 11 days, the equivalent values were 24 and 46% and ∼2.3-fold higher LDLR proteins. These data constitute a proof-of-principle for the future usage of sdAbs as PCSK9-targeting drugs that can efficiently reduce LDL-cholesterol, and as tools to study the Cys-His-rich domain-dependent sorting the PCSK9-LDLR complex to lysosomes.

摘要

单域抗体(sdAbs)对应于骆驼科动物抗体的抗原结合域。它们具有与单克隆抗体(mAbs)相同的抗原结合特性和特异性,但生产起来更容易且成本更低。我们在此报告了靶向人PCSK9(前蛋白转化酶枯草杆菌蛋白酶/kexin 9型)的sdAbs的研发情况,以此作为抗PCSK9 mAbs的替代物。用人类PCSK9免疫一只美洲驼后,我们筛选出了四种与PCSK9具有高亲和力的sdAbs,并将它们制备成与小鼠Fc的融合蛋白。所有四种sdAb-Fc均识别PCSK9的C末端富含半胱氨酸-组氨酸的结构域。我们进行了多种细胞实验,并证明所筛选的sdAbs能有效阻断PCSK9介导的细胞系、人肝细胞和小鼠原代肝细胞中低密度脂蛋白受体(LDLR)的降解。我们进一步表明,sdAb-Fc并不影响PCSK9与LDLR的结合,而是阻断其诱导的细胞内LDLR降解。构建了表达人细菌人工染色体(BAC)转基因的Pcsk9基因敲除小鼠,其血浆中人PCSK9水平约为300 ng/ml。分别对小鼠单次或双次注射最佳的sdAb-Fc,并在第4天或第11天进行分析。4天后,小鼠的总胆固醇和载脂蛋白B水平分别降低了32%和44%,肝脏LDLR蛋白水平升高了约1.8倍。在第11天,相应数值分别为24%和46%,LDLR蛋白水平升高约2.3倍。这些数据为sdAbs作为可有效降低LDL胆固醇的PCSK9靶向药物以及作为研究富含半胱氨酸-组氨酸结构域依赖性将PCSK9-LDLR复合物分选至溶酶体的工具的未来应用提供了原理证明。

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