Strekalova V V, Khachirov D G, Dedenkov A N, Suvorov Iu I, Shvatsabaia I K
Biull Vsesoiuznogo Kardiol Nauchn Tsentra AMN SSSR. 1989;12(1):48-51.
Combination of chronic salt loading with protein-poor diet produces experimental hypertension with natrium consumption near to physiological. The present model is characterized, compared to the existing one, by stage development, moderate arterial blood pressure elevation and absence of "salt toxicosis" and may be thus considered more adequate for experimental investigation of primary arterial hypertension pathophysiology.
慢性高盐饮食与低蛋白饮食相结合会导致实验性高血压,钠摄入量接近生理水平。与现有模型相比,本模型的特点是分阶段发展、动脉血压适度升高且无“盐中毒”,因此可能被认为更适合用于原发性动脉高血压病理生理学的实验研究。