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遗传易感性预测皮肤癣病。

Genetic Predictors of Susceptibility to Dermatophytoses.

机构信息

UMKC School of Medicine, Kansas City, MO, USA.

Section of Therapeutic Innovation, Children's Mercy Hospital, Kansas City, MO, USA.

出版信息

Mycopathologia. 2017 Feb;182(1-2):67-76. doi: 10.1007/s11046-016-0046-z. Epub 2016 Aug 8.

DOI:10.1007/s11046-016-0046-z
PMID:27502504
Abstract

Countless observational studies conducted over the last century reveal that dermatophytes infect humans of every age, race, gender, and socioeconomic status with strikingly high rates. The curious disparity in dermatophyte infection patterns observed within and between populations has led countless investigators to explore whether genetics underlie a susceptibility to, or confer protection against, dermatophyte infections. This paper examines the data that offer a link between genetics and dermatophytoses and discusses the underlying mechanisms that support these observations.

摘要

在上个世纪进行的无数观察性研究表明，皮肤癣菌以极高的感染率感染着每个年龄段、种族、性别和社会经济地位的人类。在人群内部和人群之间观察到的皮肤癣菌感染模式的奇特差异，促使无数研究人员探索遗传因素是否是导致皮肤癣菌感染易感性或保护作用的基础。本文探讨了遗传学与皮肤癣病之间关联的相关数据，并讨论了支持这些观察结果的潜在机制。

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本文引用的文献

[1]
A STAT1-gain-of-function mutation causing Th17 deficiency with chronic mucocutaneous candidiasis, psoriasiform hyperkeratosis and dermatophytosis.

BMJ Case Rep. 2015-10-22

[2]
IL-17 T cells' defective differentiation in vitro despite normal range ex vivo in chronic mucocutaneous candidiasis due to STAT1 mutation.

J Invest Dermatol. 2014-4

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N Engl J Med. 2013-10-16

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J Innate Immun. 2014

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Genetic predictors of susceptibility to cutaneous fungal infections: a pilot genome wide association study to refine a candidate gene search.

J Dermatol Sci. 2012-5-27

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Mendelian traits causing susceptibility to mucocutaneous fungal infections in human subjects.

J Allergy Clin Immunol. 2012-2

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PLoS One. 2011-12-14

[9]
The biological significance of TLR3 variant, L412F, in conferring susceptibility to cutaneous candidiasis, CMV and autoimmunity.

Autoimmun Rev. 2011-10-17

[10]
Autoimmune regulator (AIRE) contributes to Dectin-1-induced TNF-α production and complexes with caspase recruitment domain-containing protein 9 (CARD9), spleen tyrosine kinase (Syk), and Dectin-1.

J Allergy Clin Immunol. 2011-9-29

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