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Retraction: ER stress is associated with dedifferentiation and an epithelial-to-mesenchymal transition-like phenotype in PC Cl3 thyroid cells.

作者信息

Ulianich Luca, Garbi Corrado, Treglia Antonella Sonia, Punzi Dario, Miele Claudia, Raciti Gregory Alexander, Beguinot Francesco, Consiglio Eduardo, Di Jeso Bruno

出版信息

J Cell Sci. 2016 Sep 15;129(18):3518. doi: 10.1242/jcs.196584.

DOI:10.1242/jcs.196584
PMID:27635067
Abstract
摘要

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Retraction: ER stress is associated with dedifferentiation and an epithelial-to-mesenchymal transition-like phenotype in PC Cl3 thyroid cells.撤回声明:内质网应激与PC Cl3甲状腺细胞的去分化及上皮-间质转化样表型相关。
J Cell Sci. 2016 Sep 15;129(18):3518. doi: 10.1242/jcs.196584.
2
ER stress is associated with dedifferentiation and an epithelial-to-mesenchymal transition-like phenotype in PC Cl3 thyroid cells.内质网应激与PC Cl3甲状腺细胞的去分化及上皮-间质转化样表型相关。
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Hydrogen sulfide suppresses endoplasmic reticulum stress-induced endothelial-to-mesenchymal transition through Src pathway.硫化氢通过Src 通路抑制内质网应激诱导的内皮细胞向间充质细胞转化。
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引用本文的文献

1
The Pervasive Effects of ER Stress on a Typical Endocrine Cell: Dedifferentiation, Mesenchymal Shift and Antioxidant Response in the Thyrocyte.内质网应激对典型内分泌细胞的普遍影响:甲状腺细胞的去分化、间质转化和抗氧化反应。
Front Endocrinol (Lausanne). 2020 Nov 9;11:588685. doi: 10.3389/fendo.2020.588685. eCollection 2020.
2
The Role of the ER-Induced UPR Pathway and the Efficacy of Its Inhibitors and Inducers in the Inhibition of Tumor Progression.内质网应激诱导的 UPR 通路的作用及其抑制剂和诱导剂在抑制肿瘤进展中的疗效。
Oxid Med Cell Longev. 2019 Feb 3;2019:5729710. doi: 10.1155/2019/5729710. eCollection 2019.