Janssen T, Steinhoff H J, Böhnke F
Univ.-HNO-Klinik und Poliklinik rechts der Isar TU, München.
Laryngorhinootologie. 1989 Jul;68(7):379-82. doi: 10.1055/s-2007-998358.
Frequency-specific information on high tone loss in the inner ear can, and if so only indirectly, be obtained from changes in latency of the click-evoked brain stem potential (ABR). The relationship between latency increases of the Jewett-wave-V in basocochlear hearing loss and the time course of the travelling wave on the basilar membrane will be presented. The latency increases of basocochlear hearing loss at 2 kHz, 1 kHz and 500 Hz correspond to both the computer-simulated time course of the travelling wave and to those of the derived responses. From a pathophysiological standpoint, receptor cells in basocochlear hearing loss are not functional, beginning at the oval window, so that, dependent on the degree of inactivity, a delay corresponding to that of the time course of the travelling wave passes until active hair cells are reached and action potentials released that produce, when summed up a potential of delayed latency.
内耳高音调损失的频率特异性信息可以从点击诱发脑干电位（ABR）潜伏期的变化中获得，若能获得则也只是间接获得。将介绍基底耳蜗性听力损失中朱厄特波V潜伏期增加与基底膜上行波时间进程之间的关系。2kHz、1kHz和500Hz处基底耳蜗性听力损失的潜伏期增加既与计算机模拟的行波时间进程相对应，也与衍生反应的时间进程相对应。从病理生理学角度来看，基底耳蜗性听力损失中的感受器细胞从卵圆窗开始就没有功能，因此，根据不活动程度，直到到达活跃的毛细胞并释放动作电位，才会经过与行波时间进程相对应的延迟，这些动作电位汇总后会产生潜伏期延迟的电位。
