Somer Deniz, Cinar Fatma Gul, Kaderli Ahmet, Ornek Firdevs
Department of Pediatric Ophthalmology and Strabismus, Ankara Education and Research Hospital, Ankara, Turkey.
Department of Pediatric Ophthalmology and Strabismus, Ankara Education and Research Hospital, Ankara, Turkey.
J AAPOS. 2016 Oct;20(5):410-414.e3. doi: 10.1016/j.jaapos.2016.07.222. Epub 2016 Sep 17.
To discuss surgical intervention strategies among patients with horizontal gaze palsy with concurrent esotropia.
Five consecutive patients with dorsal pontine lesions are presented. Each patient had horizontal gaze palsy with symptomatic diplopia as a consequence of esotropia in primary gaze and an anomalous head turn to attain single binocular vision.
Clinical findings in the first 2 patients led us to presume there was complete loss of rectus muscle function from rectus muscle palsy. Based on this assumption, medial rectus recessions with simultaneous partial vertical muscle transposition (VRT) on the ipsilateral eye of the gaze palsy and recession-resection surgery on the contralateral eye were performed, resulting in significant motility limitation. Sequential recession-resection surgery without simultaneous VRT on the 3rd patient created an unexpected motility improvement to the side of gaze palsy, an observation differentiating rectus muscle palsy from paresis. Recession combined with VRT approach in the esotropic eye was abandoned on subsequent patients. Simultaneous recession-resection surgery without VRT in the next 2 patients resulted in alleviation of head postures, resolution of esotropia, and also substantial motility improvements to the ipsilateral hemifield of gaze palsy without limitations in adduction and vertical deviations.
Ocular misalignment and abnormal head posture as a result of conjugate gaze palsy can be successfully treated by basic recession-resection surgery, with the advantage of increasing versions to the ipsilateral side of the gaze palsy. Improved motility after surgery presumably represents paresis, not "paralysis," with residual innervation in rectus muscles.
探讨伴有共同性内斜视的水平凝视麻痹患者的手术干预策略。
报告5例连续的脑桥背侧病变患者。每位患者均有水平凝视麻痹,在第一眼位时因内斜视出现症状性复视,并伴有异常头位以获得双眼单视。
前2例患者的临床表现使我们推测直肌麻痹导致直肌功能完全丧失。基于这一假设,对凝视麻痹同侧眼行内直肌后徙术并同时进行部分垂直肌移位术(VRT),对侧眼行后徙-切除术,结果导致明显的眼球运动受限。第3例患者未同时行VRT的序贯后徙-切除术意外地使凝视麻痹侧的眼球运动得到改善,这一观察结果将直肌麻痹与轻瘫区分开来。随后的患者放弃了在内斜视眼中联合VRT的后徙术。接下来的2例患者未行VRT的同时后徙-切除术减轻了头位,矫正了内斜视,并且凝视麻痹同侧半视野的眼球运动也有显著改善,内收和垂直偏斜无受限。
共轭性凝视麻痹导致的眼位偏斜和异常头位可通过基本的后徙-切除术成功治疗,其优点是增加凝视麻痹同侧的同向运动。术后眼球运动改善可能代表轻瘫而非“麻痹”,直肌仍有残余神经支配。