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本文引用的文献

1
Impaired B-cell differentiation and T-cell regulatory function in four patients with Bloom's syndrome.
Clin Immunol Immunopathol. 1982 Feb;22(2):247-58. doi: 10.1016/0090-1229(82)90041-1.
2
Elevated spontaneous mutation rate in Bloom syndrome fibroblasts.布卢姆综合征成纤维细胞中自发突变率升高。
Proc Natl Acad Sci U S A. 1981 May;78(5):3133-7. doi: 10.1073/pnas.78.5.3133.
3
Bloom's syndrome: evidence for an increased mutation frequency in vivo.布卢姆综合征:体内突变频率增加的证据。
Science. 1983 Aug 26;221(4613):851-3. doi: 10.1126/science.6879180.
4
Bloom's syndrome XI. Progress report for 1983.布卢姆综合征XI。1983年进展报告。
Clin Genet. 1984 Feb;25(2):166-74. doi: 10.1111/j.1399-0004.1984.tb00480.x.
5
High mutation frequency in DNA transfected into mammalian cells.转染到哺乳动物细胞中的DNA具有高突变频率。
Proc Natl Acad Sci U S A. 1983 May;80(10):3015-9. doi: 10.1073/pnas.80.10.3015.
6
Mutagenesis of a shuttle vector plasmid in mammalian cells.穿梭载体质粒在哺乳动物细胞中的诱变
Mol Cell Biol. 1984 Mar;4(3):435-41. doi: 10.1128/mcb.4.3.435-441.1984.
7
How damaged is the biologically active subpopulation of transfected DNA?转染DNA的生物活性亚群受损程度如何?
Mol Cell Biol. 1984 Mar;4(3):387-98. doi: 10.1128/mcb.4.3.387-398.1984.
8
Somatic cells efficiently join unrelated DNA segments end-to-end.体细胞能有效地将不相关的DNA片段首尾相连。
Mol Cell Biol. 1982 Oct;2(10):1258-69. doi: 10.1128/mcb.2.10.1258-1269.1982.
9
Recombination in SV40-infected cells: viral DNA sequences at sites of circularization of transfecting linear DNA.SV40感染细胞中的重组:转染线性DNA环化位点处的病毒DNA序列
Virology. 1981 Mar;109(2):353-65. doi: 10.1016/0042-6822(81)90506-7.
10
Impaired natural killer cell activity in Bloom's syndrome could be restored by human recombinant IL-2 in vitro.
Clin Immunol Immunopathol. 1985 May;35(2):226-33. doi: 10.1016/0090-1229(85)90068-6.

在布卢姆综合征细胞中,线性质粒DNA的连接减少且容易出错。

Joining of linear plasmid DNA is reduced and error-prone in Bloom's syndrome cells.

作者信息

Rünger T M, Kraemer K H

机构信息

Laboratory of Molecular Carcinogenesis, National Cancer Institute, Bethesda, MD 20892.

出版信息

EMBO J. 1989 May;8(5):1419-25. doi: 10.1002/j.1460-2075.1989.tb03523.x.

DOI:10.1002/j.1460-2075.1989.tb03523.x
PMID:2767047
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC400969/
Abstract

A linearized, replicating, shuttle vector plasmid, pZ189, was used to measure in vivo DNA joining ability of cells from patients with the cancer-prone, immunodeficient, chromosome breakage disorder, Bloom's syndrome (BS). The BS cell lines we studied were reported to contain reduced in vitro activity of DNA ligase I. We assessed in vivo joining ability by transfecting linear plasmids with overlapping or blunt ends (produced by EcoRI or StuI) into BS and normal fibroblast or lymphoblast host cells and measuring the amount of re-joined, replicated plasmids by their ability to transform bacteria. With plasmids having either overlapping or blunt ends we found a 1.3- to 3-fold lower (P less than 0.05) joining efficiency in BS cells than in the normal cells. The mutation frequency of the recovered plasmids was measured by screening for function of the suppressor tRNA contained in pZ189, for plasmid size, for presence of restriction sites, or by DNA sequencing. The spontaneous mutation frequency with the circular plasmid was 0.05-0.08% with both BS cell lines, values 2- to 21-fold higher (P less than 0.03) than with the normal cell lines. The mutation frequency with the linear plasmid passaged through both BS cell lines was 21-52%, values 1.4- to 5.4-fold higher (P less than 0.001) than with the normal lines. Detailed analysis of 210 recovered plasmids revealed an increase (P less than or equal to 0.001) in deletions, insertions or complex mutations at the joining sites, and in point mutations with the EcoRI cut plasmid with the BS cells in comparison to the normal cells.(ABSTRACT TRUNCATED AT 250 WORDS)

摘要

一种线性化、可复制的穿梭载体质粒pZ189,被用于检测来自患有癌症倾向、免疫缺陷、染色体断裂疾病——布卢姆综合征(BS)患者细胞的体内DNA连接能力。我们研究的BS细胞系据报道含有降低的DNA连接酶I体外活性。我们通过将具有重叠或平端(由EcoRI或StuI产生)的线性质粒转染到BS和正常成纤维细胞或淋巴母细胞宿主细胞中,并通过其转化细菌的能力来测量重新连接、复制的质粒数量,从而评估体内连接能力。对于具有重叠或平端的质粒,我们发现BS细胞中的连接效率比正常细胞低1.3至3倍(P小于0.05)。通过筛选pZ189中所含抑制性tRNA的功能、质粒大小、限制酶切位点的存在情况或通过DNA测序来测量回收质粒的突变频率。两种BS细胞系中环状质粒的自发突变频率为0.05 - 0.08%,比正常细胞系高2至21倍(P小于0.03)。通过两种BS细胞系传代的线性质粒的突变频率为21 - 52%,比正常细胞系高1.4至5.4倍(P小于0.001)。对210个回收质粒的详细分析显示,与正常细胞相比,BS细胞中连接位点处的缺失、插入或复杂突变以及EcoRI切割质粒的点突变有所增加(P小于或等于0.001)。(摘要截短于250字)