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Arachidonic acid metabolism in ischemic neuronal damage.

作者信息

Abe K, Yoshidomi M, Kogure K

机构信息

Department of Neurology, Tohoku University School of Medicine, Sendai, Japan.

出版信息

Ann N Y Acad Sci. 1989;559:259-68. doi: 10.1111/j.1749-6632.1989.tb22614.x.

DOI:10.1111/j.1749-6632.1989.tb22614.x
PMID:2774401
Abstract
摘要

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Arachidonic acid metabolism in ischemic neuronal damage.缺血性神经元损伤中的花生四烯酸代谢
Ann N Y Acad Sci. 1989;559:259-68. doi: 10.1111/j.1749-6632.1989.tb22614.x.
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Arachidonic acid metabolism and cerebral blood flow in the normal, ischemic, and reperfused gerbil brain. Inhibition of ischemia-reperfusion-induced cerebral injury by a platelet-activating factor antagonist (BN 52021).正常、缺血及再灌注沙土鼠脑内的花生四烯酸代谢与脑血流量。血小板活化因子拮抗剂（BN 52021）对缺血再灌注诱导的脑损伤的抑制作用。
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Transient modifications in brain free arachidonic acid in experimental animals durng convulsions.实验动物惊厥期间脑游离花生四烯酸的短暂变化。
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The role of early Ca2+ influx in the pathogenesis of delayed neuronal death after brief forebrain ischemia in gerbils.早期钙离子内流在沙鼠短暂前脑缺血后迟发性神经元死亡发病机制中的作用。
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Effect of transient ischemia on free fatty acids and phospholipids in the gerbil brain. Lipid peroxidation as a possible cause of postischemic injury.短暂性缺血对沙鼠脑游离脂肪酸和磷脂的影响。脂质过氧化作为缺血后损伤的可能原因。
J Neurosurg. 1980 Sep;53(3):323-31. doi: 10.3171/jns.1980.53.3.0323.

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Palmitic and stearic fatty acids induce caspase-dependent and -independent cell death in nerve growth factor differentiated PC12 cells.棕榈酸和硬脂酸在神经生长因子分化的PC12细胞中诱导半胱天冬酶依赖性和非依赖性细胞死亡。
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