• 文献检索
  • 文档翻译
  • 深度研究
  • 学术资讯
  • Suppr Zotero 插件Zotero 插件
  • 邀请有礼
  • 套餐&价格
  • 历史记录
应用&插件
Suppr Zotero 插件Zotero 插件浏览器插件Mac 客户端Windows 客户端微信小程序
定价
高级版会员购买积分包购买API积分包
服务
文献检索文档翻译深度研究API 文档MCP 服务
关于我们
关于 Suppr公司介绍联系我们用户协议隐私条款
关注我们

Suppr 超能文献

核心技术专利：CN118964589B侵权必究
粤ICP备2023148730 号-1Suppr @ 2026

文献检索

告别复杂PubMed语法，用中文像聊天一样搜索，搜遍4000万医学文献。AI智能推荐，让科研检索更轻松。

立即免费搜索

文件翻译

保留排版，准确专业，支持PDF/Word/PPT等文件格式，支持 12+语言互译。

免费翻译文档

深度研究

AI帮你快速写综述，25分钟生成高质量综述，智能提取关键信息，辅助科研写作。

立即免费体验

Pericytes Preserve Capillary Integrity to Prevent Kidney Hypoxia.

作者信息

Venkatachalam Manjeri A, Weinberg Joel M

机构信息

Department of Pathology, University of Texas Health Science Center, San Antonio, Texas; and

Department of Medicine, University of Michigan Medical Center, Ann Arbor, Michigan.

出版信息

J Am Soc Nephrol. 2017 Mar;28(3):717-719. doi: 10.1681/ASN.2016111157. Epub 2016 Dec 15.

DOI:10.1681/ASN.2016111157
PMID:27979991
原文链接:https://pmc.ncbi.nlm.nih.gov/articles/PMC5328172/
Abstract
摘要

相似文献

1
Pericytes Preserve Capillary Integrity to Prevent Kidney Hypoxia.周细胞维持毛细血管完整性以预防肾脏缺氧。
J Am Soc Nephrol. 2017 Mar;28(3):717-719. doi: 10.1681/ASN.2016111157. Epub 2016 Dec 15.
2
Gli1 Pericyte Loss Induces Capillary Rarefaction and Proximal Tubular Injury.Gli1+周细胞缺失诱导毛细血管稀疏和近端肾小管损伤。
J Am Soc Nephrol. 2017 Mar;28(3):776-784. doi: 10.1681/ASN.2016030297. Epub 2016 Sep 13.
3
Peritubular capillary loss is ameliorated by ramipril or valsartan treatment.
Microcirculation. 2008 May;15(4):337-48. doi: 10.1080/10739680701743540.
4
Regardless of etiology, progressive renal disease causes ultrastructural and functional alterations of peritubular capillaries.无论病因如何，进行性肾病都会导致肾小管周围毛细血管的超微结构和功能改变。
Kidney Int. 2017 Jan;91(1):70-85. doi: 10.1016/j.kint.2016.07.038. Epub 2016 Sep 24.
5
[ON THE PATHOGENESIS OF THE SHOCK KIDNEY].[论休克肾的发病机制]
Munch Med Wochenschr. 1964 May 22;106:982-4.
6
Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes.慢性肾脏病中的缺氧与纤维化：在周细胞处交汇
Kidney Int Suppl (2011). 2014 Nov;4(1):107-112. doi: 10.1038/kisup.2014.20.
7
Dysmorphogenesis of kidney cortical peritubular capillaries in angiopoietin-2-deficient mice.血管生成素-2缺陷小鼠肾皮质肾小管周围毛细血管的畸形发生
Am J Pathol. 2004 Dec;165(6):1895-906. doi: 10.1016/S0002-9440(10)63242-7.
8
Intravital videomicroscopy of peritubular capillaries in renal ischemia.肾缺血时肾小管周围毛细血管的活体视频显微镜检查
Am J Physiol Renal Physiol. 2002 Jun;282(6):F1150-5. doi: 10.1152/ajprenal.00310.2001.
9
Peritubular capillaries: an important piece of the puzzle.肾小管周毛细血管：拼图的重要一块。
Kidney Int. 2017 Jan;91(1):9-11. doi: 10.1016/j.kint.2016.08.033.
10
Peritubular capillary preservation with COMP-angiopoietin-1 decreases ischemia-reperfusion-induced acute kidney injury.使用COMP-血管生成素-1保存肾小管周围毛细血管可减少缺血再灌注诱导的急性肾损伤。
Am J Physiol Renal Physiol. 2009 Oct;297(4):F952-60. doi: 10.1152/ajprenal.00064.2009. Epub 2009 Aug 5.

引用本文的文献

1
Molecular mechanisms and therapeutic advances of peritubular capillary neogenesis in acute kidney injury.急性肾损伤时肾小管周围毛细血管新生的分子机制与治疗进展
Front Mol Biosci. 2025 Aug 20;12:1643838. doi: 10.3389/fmolb.2025.1643838. eCollection 2025.
2
Bioengineered platelet nanoplatform enables renal-targeted dexamethasone delivery for chronic nephritis therapy with dual anti-inflammatory/anti-fibrotic effects and minimized systemic toxicity.生物工程血小板纳米平台实现肾靶向地塞米松递送，用于慢性肾炎治疗，具有双重抗炎/抗纤维化作用，并将全身毒性降至最低。
Bioact Mater. 2025 Jun 7;52:213-227. doi: 10.1016/j.bioactmat.2025.06.002. eCollection 2025 Oct.
3
Aging, vascular dysfunction, and the blood-brain barrier: unveiling the pathophysiology of stroke in older adults.衰老、血管功能障碍与血脑屏障：揭示老年人中风的病理生理学
Biogerontology. 2025 Mar 6;26(2):67. doi: 10.1007/s10522-025-10209-y.
4
Modulation of gentamicin-induced acute kidney injury by myo-inositol oxygenase via the ROS/ALOX-12/12-HETE/GPR31 signaling pathway.肌醇加双氧酶通过 ROS/ALOX-12/12-HETE/GPR31 信号通路调节庆大霉素诱导的急性肾损伤。
JCI Insight. 2022 Mar 22;7(6):e155487. doi: 10.1172/jci.insight.155487.
5
Exogenous bone marrow derived-putative endothelial progenitor cells attenuate ischemia reperfusion-induced vascular injury and renal fibrosis in mice dependent on pericytes.外源性骨髓来源的拟血管内皮祖细胞通过周细胞依赖途径减轻小鼠缺血再灌注诱导的血管损伤和肾纤维化。
Theranostics. 2020 Oct 25;10(26):12144-12157. doi: 10.7150/thno.48562. eCollection 2020.
6
Adjunctive mesenchymal stem/stromal cells augment microvascular function in poststenotic kidneys treated with low-energy shockwave therapy.低能量冲击波治疗后狭窄肾脏中辅助间充质干细胞/基质细胞增强微血管功能。
J Cell Physiol. 2020 Dec;235(12):9806-9818. doi: 10.1002/jcp.29794. Epub 2020 May 19.
7
Pericytes in Cerebrovascular Diseases: An Emerging Therapeutic Target.脑血管疾病中的周细胞：一个新兴的治疗靶点
Front Cell Neurosci. 2019 Nov 22;13:519. doi: 10.3389/fncel.2019.00519. eCollection 2019.
8
Putative endothelial progenitor cells do not promote vascular repair but attenuate pericyte-myofibroblast transition in UUO-induced renal fibrosis.推定的内皮祖细胞不能促进血管修复，但可减轻 UUO 诱导的肾脏纤维化中的周细胞-肌成纤维细胞转化。
Stem Cell Res Ther. 2019 Mar 21;10(1):104. doi: 10.1186/s13287-019-1201-5.

本文引用的文献

1
Gli1 Pericyte Loss Induces Capillary Rarefaction and Proximal Tubular Injury.Gli1+周细胞缺失诱导毛细血管稀疏和近端肾小管损伤。
J Am Soc Nephrol. 2017 Mar;28(3):776-784. doi: 10.1681/ASN.2016030297. Epub 2016 Sep 13.
2
Failed Tubule Recovery, AKI-CKD Transition, and Kidney Disease Progression.肾小管修复失败、急性肾损伤-慢性肾病转化及肾病进展。
J Am Soc Nephrol. 2015 Aug;26(8):1765-76. doi: 10.1681/ASN.2015010006. Epub 2015 Mar 25.
3
Perivascular Gli1+ progenitors are key contributors to injury-induced organ fibrosis.血管周围Gli1+祖细胞是损伤诱导器官纤维化的关键促成因素。
Cell Stem Cell. 2015 Jan 8;16(1):51-66. doi: 10.1016/j.stem.2014.11.004. Epub 2014 Nov 20.
4
The chronic hypoxia hypothesis: the search for the smoking gun goes on.慢性缺氧假说：寻找确凿证据的工作仍在继续。
Am J Physiol Renal Physiol. 2015 Jan 15;308(2):F101-2. doi: 10.1152/ajprenal.00587.2014. Epub 2014 Nov 5.
5
Hypoxia as a key player in the AKI-to-CKD transition.缺氧在急性肾损伤向慢性肾脏病转变过程中起关键作用。
Am J Physiol Renal Physiol. 2014 Dec 1;307(11):F1187-95. doi: 10.1152/ajprenal.00425.2014. Epub 2014 Oct 1.
6
Renal oxygenation in acute renal ischemia-reperfusion injury.急性肾缺血再灌注损伤中的肾脏氧合。
Am J Physiol Renal Physiol. 2014 May 1;306(9):F1026-38. doi: 10.1152/ajprenal.00281.2013. Epub 2014 Mar 5.
7
EphrinB2 reverse signaling protects against capillary rarefaction and fibrosis after kidney injury.EphrinB2 反向信号转导可预防肾损伤后的毛细血管稀疏和纤维化。
J Am Soc Nephrol. 2013 Mar;24(4):559-72. doi: 10.1681/ASN.2012080871. Epub 2013 Mar 14.
8
Targeting endothelium-pericyte cross talk by inhibiting VEGF receptor signaling attenuates kidney microvascular rarefaction and fibrosis.靶向血管内皮细胞-周细胞相互作用抑制 VEGF 受体信号通路可减轻肾脏微血管稀疏和纤维化。
Am J Pathol. 2011 Feb;178(2):911-23. doi: 10.1016/j.ajpath.2010.10.012.
9
Cell respiration under hypoxia: facts and artefacts in mitochondrial oxygen kinetics.低氧条件下的细胞呼吸：线粒体氧动力学的事实和假象。
Adv Exp Med Biol. 2010;662:7-25. doi: 10.1007/978-1-4419-1241-1_2.
10
Pimonidazole adduct immunohistochemistry in the rat kidney: detection of tissue hypoxia.大鼠肾脏中匹莫硝唑加合物免疫组织化学：组织缺氧的检测
Methods Mol Biol. 2009;466:161-74. doi: 10.1007/978-1-59745-352-3_12.