How ICE lights the pyroptosis fire.
作者信息
Liu Xing, Lieberman Judy
机构信息
Program in Cellular and Molecular Medicine Children's Hospital and Department of Pediatrics, Harvard Medical School, Boston, MA, USA.
出版信息
Cell Death Differ. 2017 Feb;24(2):197-199. doi: 10.1038/cdd.2016.157. Epub 2017 Jan 6.
Abstract
摘要
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Growth inhibition of cytosolic Salmonella by caspase-1 and caspase-11 precedes host cell death.细胞质沙门氏菌的生长抑制由胱天蛋白酶-1 和胱天蛋白酶-11 引起，早于宿主细胞死亡。
Nat Commun. 2016 Nov 3;7:13292. doi: 10.1038/ncomms13292.
2
GSDMB induces an asthma phenotype characterized by increased airway responsiveness and remodeling without lung inflammation.Gasdermin B（GSDMB）诱导出一种哮喘表型，其特征为气道反应性增加和重塑，而无肺部炎症。
Proc Natl Acad Sci U S A. 2016 Nov 15;113(46):13132-13137. doi: 10.1073/pnas.1610433113. Epub 2016 Oct 31.
3
Pyroptosis is driven by non-selective gasdermin-D pore and its morphology is different from MLKL channel-mediated necroptosis.细胞焦亡由非选择性的gasdermin-D孔道驱动，其形态不同于MLKL通道介导的坏死性凋亡。
Cell Res. 2016 Sep;26(9):1007-20. doi: 10.1038/cr.2016.100. Epub 2016 Aug 30.
4
GSDMD membrane pore formation constitutes the mechanism of pyroptotic cell death.Gasdermin D（GSDMD）膜孔形成构成了细胞焦亡性细胞死亡的机制。
EMBO J. 2016 Aug 15;35(16):1766-78. doi: 10.15252/embj.201694696. Epub 2016 Jul 14.
5
Inflammasome-activated gasdermin D causes pyroptosis by forming membrane pores.炎性小体激活的gasdermin D通过形成膜孔导致细胞焦亡。
Nature. 2016 Jul 7;535(7610):153-8. doi: 10.1038/nature18629.
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GsdmD p30 elicited by caspase-11 during pyroptosis forms pores in membranes.细胞焦亡过程中由半胱天冬酶-11诱导产生的Gasdermin D p30在细胞膜上形成孔道。
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Pore-forming activity and structural autoinhibition of the gasdermin family.气 分 子 家 族 的 孔 形 成 活 性 和 结 构 自 抑 制 。
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Caspase-11 cleaves gasdermin D for non-canonical inflammasome signalling.半胱氨酸天冬氨酸蛋白酶 11 切割天冬氨酸半胱氨酸酶蛋白 D 以进行非经典炎性小体信号转导。
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Cleavage of GSDMD by inflammatory caspases determines pyroptotic cell death.炎性小体天冬氨酸特异性半胱氨酸蛋白酶 1（caspase-1）切割 GSDMD 决定细胞焦亡。
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