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The TP53 Pro72Arg SNP in de novo acute myeloid leukaemia - results of two cohort studies involving 215 patients and 3759 controls.

作者信息

Schulz Eduard, Lind Karin, Renner Wilfried, Petersen Britt-Sabina, Quehenberger Franz, Dill Claudia, Hofer Sybille, Lal Ridhima, Hoefler Gerald, Schlenke Peter, Ehninger Gerhard, Schetelig Johannes, Middeke Jan M, Stölzel Friedrich, Sill Heinz

机构信息

Division of Haematology, Department of Internal Medicine, Medical University of Graz, Graz, Austria.

Clinical Institute of Medical and Chemical Laboratory Diagnostics, Medical University of Graz, Graz, Austria.

出版信息

Br J Haematol. 2018 Apr;181(1):148-151. doi: 10.1111/bjh.14527. Epub 2017 Jan 20.

DOI:10.1111/bjh.14527
PMID:28106906
Abstract
摘要

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The TP53 Pro72Arg SNP in de novo acute myeloid leukaemia - results of two cohort studies involving 215 patients and 3759 controls.初发急性髓系白血病中TP53 Pro72Arg单核苷酸多态性——两项涉及215例患者和3759例对照的队列研究结果
Br J Haematol. 2018 Apr;181(1):148-151. doi: 10.1111/bjh.14527. Epub 2017 Jan 20.
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TP53 mutation characteristics in therapy-related myelodysplastic syndromes and acute myeloid leukemia is similar to de novo diseases.治疗相关的骨髓增生异常综合征和急性髓系白血病中的TP53突变特征与原发性疾病相似。
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Prognostic significance of TP53 mutations and single nucleotide polymorphisms in acute myeloid leukemia: a case series and literature review.TP53突变和单核苷酸多态性在急性髓系白血病中的预后意义：病例系列及文献综述
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Association between the TP53 Arg72Pro polymorphism and clinical outcomes in acute myeloid leukemia.TP53基因Arg72Pro多态性与急性髓系白血病临床结局的关联
Haematologica. 2017 Feb;102(2):e43-e46. doi: 10.3324/haematol.2016.155069. Epub 2016 Oct 20.
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TP53 mutations and MDM2(SNP309) identify subgroups of AML patients with impaired outcome.TP53突变和MDM2（SNP309）可识别预后不良的急性髓系白血病患者亚组。
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TP53 Gene Pro72Arg (rs1042522) Single Nucleotide Polymorphism as Not a Risk Factor for Colorectal Cancer in the Iranian Azari Population.TP53基因Pro72Arg（rs1042522）单核苷酸多态性并非伊朗阿扎里人群患结直肠癌的风险因素。
Asian Pac J Cancer Prev. 2017 Dec 29;18(12):3423-3427. doi: 10.22034/APJCP.2017.18.12.3423.
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Single-molecule DNA sequencing of acute myeloid leukemia and myelodysplastic syndromes with multiple alterations.伴有多种改变的急性髓系白血病和骨髓增生异常综合征的单分子DNA测序
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Overexpression of TP53 is associated with poor survival, but not with reduced response to hypomethylating agents in older patients with acute myeloid leukaemia.TP53的过表达与较差的生存率相关，但与老年急性髓系白血病患者对低甲基化药物反应降低无关。
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The TP53 Pro72Arg SNP in de novo acute myeloid leukemia.初发急性髓系白血病中的TP53 Pro72Arg单核苷酸多态性
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引用本文的文献

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Biomark Res. 2024 Nov 14;12(1):137. doi: 10.1186/s40364-024-00676-9.
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Germ line variant GFI1-36N affects DNA repair and sensitizes AML cells to DNA damage and repair therapy.胚系变异 GFI1-36N 影响 DNA 修复，并使 AML 细胞对 DNA 损伤和修复治疗敏感。
Blood. 2023 Dec 21;142(25):2175-2191. doi: 10.1182/blood.2022015752.
3
The TP53 Pro72Arg SNP in de novo acute myeloid leukemia.
初发急性髓系白血病中的TP53 Pro72Arg单核苷酸多态性
Haematologica. 2017 May;102(5):e214-e215. doi: 10.3324/haematol.2017.165019.