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Susceptibility to mouse hepatitis virus strain 3 in BALB/cJ mice: failure of immune cell proliferation and interleukin 2 production.

作者信息

Dindzans V J, Zimmerman B, Sherker A, Levy G A

机构信息

Mount Sinai Hospital, University of Toronto, Ontario, Canada.

出版信息

Adv Exp Med Biol. 1987;218:411-20. doi: 10.1007/978-1-4684-1280-2_50.

DOI:10.1007/978-1-4684-1280-2_50
PMID:2829553
Abstract
摘要

相似文献

1
Susceptibility to mouse hepatitis virus strain 3 in BALB/cJ mice: failure of immune cell proliferation and interleukin 2 production.BALB/cJ小鼠对3型小鼠肝炎病毒的易感性:免疫细胞增殖和白细胞介素2产生的失败
Adv Exp Med Biol. 1987;218:411-20. doi: 10.1007/978-1-4684-1280-2_50.
2
Infection of BALB/cByJ mice with the JHM strain of mouse hepatitis virus alters in vitro splenic T cell proliferation and cytokine production.用小鼠肝炎病毒JHM株感染BALB/cByJ小鼠会改变体外脾T细胞增殖和细胞因子产生。
Lab Anim Sci. 1991 Apr;41(2):99-105.
3
In vitro splenic T cell responses of diverse mouse genotypes after oronasal exposure to mouse hepatitis virus, strain JHM.
Lab Anim Sci. 1991 Apr;41(2):106-11.
4
Biological properties of interleukins.
Allergy. 1983 May;38(4):219-26. doi: 10.1111/j.1398-9995.1983.tb01614.x.
5
Induction of receptors for interleukin 2 requires T cell Ag:Ia receptor crosslinking and interleukin 1.白细胞介素2受体的诱导需要T细胞抗原:Ia受体交联和白细胞介素1。
Lymphokine Res. 1984 Summer;3(4):175-82.
6
Resistance of naive mice to murine hepatitis virus strain 3 requires development of a Th1, but not a Th2, response, whereas pre-existing antibody partially protects against primary infection.未接触过抗原的小鼠对鼠肝炎病毒3型的抵抗力需要Th1应答的产生,而非Th2应答,而预先存在的抗体可部分保护小鼠免受初次感染。
J Immunol. 1996 May 1;156(9):3342-9.
7
Interleukins and the immune system 1.白细胞介素与免疫系统1。
Lancet. 1989 Apr 29;1(8644):943-7.
8
Spontaneous production of interleukin-2 and interleukin-3 by spleen cells from mice infected with mouse hepatitis virus type 4.感染4型小鼠肝炎病毒的小鼠脾脏细胞自发产生白细胞介素-2和白细胞介素-3 。
J Virol. 1988 Sep;62(9):3506-8. doi: 10.1128/JVI.62.9.3506-3508.1988.
9
Ionophore A23-187 induces responses to TCGF in mouse lymphocytes.离子载体A23 - 187可诱导小鼠淋巴细胞对TCGF产生反应。
Ann Immunol (Paris). 1984 May-Jun;135C(3):365-73. doi: 10.1016/s0769-2625(84)80966-6.
10
Regulation of the immune response T-cell interactions.免疫反应的调节:T细胞相互作用
CRC Crit Rev Immunol. 1982 Feb;3(3):209-61.

引用本文的文献

1
Multi-Organ Histopathological Changes in a Mouse Hepatitis Virus Model of COVID-19.新型冠状病毒肺炎小鼠肝炎病毒模型中的多器官组织病理变化。
Viruses. 2021 Aug 27;13(9):1703. doi: 10.3390/v13091703.
2
Murine hepatitis virus strain 1 produces a clinically relevant model of severe acute respiratory syndrome in A/J mice.鼠肝炎病毒1型在A/J小鼠中产生了一种与严重急性呼吸综合征临床相关的模型。
J Virol. 2006 Nov;80(21):10382-94. doi: 10.1128/JVI.00747-06.
3
Interference of natural mouse hepatitis virus infection with cytokine production and susceptibility to Trypanosoma cruzi.
自然小鼠肝炎病毒感染对细胞因子产生及克氏锥虫易感性的干扰
Immunology. 1999 Mar;96(3):381-8. doi: 10.1046/j.1365-2567.1999.00719.x.
4
Immunodepression induced by Trypanosoma cruzi and mouse hepatitis virus type 3 is associated with thymus apoptosis.克氏锥虫和3型小鼠肝炎病毒诱导的免疫抑制与胸腺细胞凋亡有关。
Clin Diagn Lab Immunol. 1998 Mar;5(2):186-91. doi: 10.1128/CDLI.5.2.186-191.1998.
5
A Th1 cell line (3E9.1) from resistant A/J mice inhibits induction of macrophage procoagulant activity in vitro and protects against MHV-3 mortality in vivo.来自抗性A/J小鼠的Th1细胞系(3E9.1)在体外可抑制巨噬细胞促凝活性的诱导,并在体内预防MHV-3致死。
Immunology. 1994 Nov;83(3):353-61.
6
Pathogenicity of neutralization escape mutants of mouse hepatitis virus: correlation with T- and B-cell depletions.小鼠肝炎病毒中和逃逸突变体的致病性:与T细胞和B细胞耗竭的相关性。
Res Immunol. 1994 Sep;145(7):553-65. doi: 10.1016/s0923-2494(94)80073-1.
7
Pattern of disease after murine hepatitis virus strain 3 infection correlates with macrophage activation and not viral replication.鼠肝炎病毒3型感染后的疾病模式与巨噬细胞激活相关,而非病毒复制。
J Virol. 1995 Sep;69(9):5252-60. doi: 10.1128/JVI.69.9.5252-5260.1995.
8
Monoclonal antiprothrombinase (3D4.3) prevents mortality from murine hepatitis virus (MHV-3) infection.单克隆抗凝血酶原酶(3D4.3)可预防小鼠感染鼠肝炎病毒(MHV-3)后的死亡。
J Exp Med. 1992 Sep 1;176(3):689-97. doi: 10.1084/jem.176.3.689.