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DUX4-induced constitutive DNA damage and oxidative stress contribute to aberrant differentiation of myoblasts from FSHD patients.
Free Radic Biol Med. 2016 Oct;99:244-258. doi: 10.1016/j.freeradbiomed.2016.08.007. Epub 2016 Aug 9.
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Temporal activation of XRCC1-mediated DNA repair is essential for muscle differentiation.
Cell Discov. 2016 Jan 12;2:15041. doi: 10.1038/celldisc.2015.41. eCollection 2016.
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Pathways controlling dNTP pools to maintain genome stability.
DNA Repair (Amst). 2016 Aug;44:193-204. doi: 10.1016/j.dnarep.2016.05.032. Epub 2016 May 28.
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Facioscapulohumeral dystrophy myoblasts efficiently repair moderate levels of oxidative DNA damage.
Histochem Cell Biol. 2016 Apr;145(4):475-83. doi: 10.1007/s00418-016-1410-2. Epub 2016 Feb 9.
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Genome Integrity in Aging: Human Syndromes, Mouse Models, and Therapeutic Options.
Annu Rev Pharmacol Toxicol. 2016;56:427-45. doi: 10.1146/annurev-pharmtox-010814-124316. Epub 2015 Oct 28.
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Apoptosis and necrosis mediate skeletal muscle fiber loss in age-induced mitochondrial enzymatic abnormalities.
Aging Cell. 2015 Dec;14(6):1085-93. doi: 10.1111/acel.12399. Epub 2015 Sep 14.
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Stress-induced DNA damage biomarkers: applications and limitations.
Front Chem. 2015 Jun 2;3:35. doi: 10.3389/fchem.2015.00035. eCollection 2015.
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Dietary restriction, mitochondrial function and aging: from yeast to humans.
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